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RJR: Recommended Bibliography 04 Sep 2026 at 01:42 Created:
Calcium Metabolism and Urinary Stones
Wikipedia: Calcium Metabolism
— everybody knows that osteoporosis is a huge threat and that the best way
to reduce the risk of osteoporosis is to take lots of dietary calcium supplements
and vitamin D pills.
OK, exercise is good, too, but that can involve actually
breaking a sweat — something that not everyone wants to do.
But, did you also know that hypercalciuria (too much secreted calcium in the urine) is
associated with a significant increase in the risk for kidney stones? And, you can
easily get hypercalciuria if you pop too many calcium-supplement pills.
Hmmmm.
Nice choice, break a hip or pass kidney stones... What to do? As with most things
biological, reality lies somewhere in the trade-off zone. A quick look at the
literature dealing with calciuria and kidney stones is one way to start.
Created with PubMed® Query: (calciuria OR "calcium metabolism") AND (urolith OR "kidney stone" OR "kidney stones" OR "bladder stones" OR stones) NOT pmcbook NOT ispreviousversion
Citations The Papers (from PubMed®)
RevDate: 2019-09-11
CmpDate: 1977-04-30
A contribution to the formation mechanism of calcium oxalate urinary calculi. II. In vitro experiments concerning the theory of the formation of Whewellite and Weddellite urinary calculi.
Urological research, 4(4):157-160.
In vitro investigations of the formation of Whewellite or Weddellite are described. By means of different precipitation models the influence of cationic minerals on the formation of Weddellite could be observed. The possible conversion of Weddellite into Whewellite in vivo is demonstrated by in vitro experiments. A theory of the formation of Weddellite or Whewellite urinary calculi is developed on the basis of the results obtained.
Additional Links: PMID-14431
PubMed:
Citation:
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@article {pmid14431,
year = {1976},
author = {Hesse, A and Berg, W and Schneider, HJ and Hienzsch, E},
title = {A contribution to the formation mechanism of calcium oxalate urinary calculi. II. In vitro experiments concerning the theory of the formation of Whewellite and Weddellite urinary calculi.},
journal = {Urological research},
volume = {4},
number = {4},
pages = {157-160},
pmid = {14431},
issn = {0300-5623},
mesh = {Calcium/*metabolism ; Crystallization ; Fractional Precipitation ; Humans ; Hydrogen-Ion Concentration ; Models, Biological ; Oxalates/*metabolism ; Urinary Calculi/*metabolism ; },
abstract = {In vitro investigations of the formation of Whewellite or Weddellite are described. By means of different precipitation models the influence of cationic minerals on the formation of Weddellite could be observed. The possible conversion of Weddellite into Whewellite in vivo is demonstrated by in vitro experiments. A theory of the formation of Weddellite or Whewellite urinary calculi is developed on the basis of the results obtained.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/*metabolism
Crystallization
Fractional Precipitation
Humans
Hydrogen-Ion Concentration
Models, Biological
Oxalates/*metabolism
Urinary Calculi/*metabolism
RevDate: 2019-09-11
CmpDate: 1977-04-30
A contribution to the formation mechanism of calcium oxalate urinary calculi. III. On the role of magnesium in the formation of oxalate calculi.
Urological research, 4(4):161-167.
The influence of magnesium in vitro on the precipitation of calcium oxalate was investigated. Even at maximum physiological magnesium concentrations a litholytic effect could not be observed, but the retardation of the calcium oxalate crystallization caused by magnesium might be decisive for a reduction in calculi formation. The enlargement of the calcium oxalate crystals and aggregates caused by the retardation of crystallization, however, should be regarded as a contraindicating factor for Mg therapy in oxalate calculous disease. It is safe to say that high magnesium concentrations prevent the conversion into Whewellite of the calcium oxalate calculi substance primarily formed as Weddellite.
Additional Links: PMID-14432
PubMed:
Citation:
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@article {pmid14432,
year = {1976},
author = {Berg, W and Hesse, A and Schneider, HJ},
title = {A contribution to the formation mechanism of calcium oxalate urinary calculi. III. On the role of magnesium in the formation of oxalate calculi.},
journal = {Urological research},
volume = {4},
number = {4},
pages = {161-167},
pmid = {14432},
issn = {0300-5623},
mesh = {Calcium/*metabolism ; Crystallization ; Fractional Precipitation ; Humans ; Hydrogen-Ion Concentration ; Magnesium/*pharmacology ; Oxalates/*metabolism ; Solubility ; Urinary Calculi/*metabolism ; },
abstract = {The influence of magnesium in vitro on the precipitation of calcium oxalate was investigated. Even at maximum physiological magnesium concentrations a litholytic effect could not be observed, but the retardation of the calcium oxalate crystallization caused by magnesium might be decisive for a reduction in calculi formation. The enlargement of the calcium oxalate crystals and aggregates caused by the retardation of crystallization, however, should be regarded as a contraindicating factor for Mg therapy in oxalate calculous disease. It is safe to say that high magnesium concentrations prevent the conversion into Whewellite of the calcium oxalate calculi substance primarily formed as Weddellite.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/*metabolism
Crystallization
Fractional Precipitation
Humans
Hydrogen-Ion Concentration
Magnesium/*pharmacology
Oxalates/*metabolism
Solubility
Urinary Calculi/*metabolism
RevDate: 2013-11-21
CmpDate: 1979-09-27
Urinary calcium excretion in human beings.
The New England journal of medicine, 301(10):535-541.
Additional Links: PMID-37442
Publisher:
PubMed:
Citation:
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@article {pmid37442,
year = {1979},
author = {Bleich, HL and Moore, MJ and Lemann, J and Adams, ND and Gray, RW},
title = {Urinary calcium excretion in human beings.},
journal = {The New England journal of medicine},
volume = {301},
number = {10},
pages = {535-541},
doi = {10.1056/NEJM197909063011008},
pmid = {37442},
issn = {0028-4793},
mesh = {Acidosis/metabolism ; Adult ; Animals ; Calcium/metabolism/*urine ; Calcium Metabolism Disorders/metabolism ; Calcium, Dietary/metabolism ; Diet ; Diuretics/pharmacology ; Glomerular Filtration Rate ; Humans ; Hydrogen-Ion Concentration ; Hydroxycholecalciferols/blood ; Kidney Calculi/metabolism ; Kidney Tubules/physiology ; Phosphates/metabolism ; Sodium/metabolism ; Urinary Calculi/etiology ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Acidosis/metabolism
Adult
Animals
Calcium/metabolism/*urine
Calcium Metabolism Disorders/metabolism
Calcium, Dietary/metabolism
Diet
Diuretics/pharmacology
Glomerular Filtration Rate
Humans
Hydrogen-Ion Concentration
Hydroxycholecalciferols/blood
Kidney Calculi/metabolism
Kidney Tubules/physiology
Phosphates/metabolism
Sodium/metabolism
Urinary Calculi/etiology
RevDate: 2019-09-02
CmpDate: 1978-09-30
A microbiological study of dental calculus. II. The in vitro calcification of microorganisms from dental calculus.
Journal of periodontal research, 13(4):360-366.
Additional Links: PMID-149855
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PubMed:
Citation:
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@article {pmid149855,
year = {1978},
author = {Sidaway, DA},
title = {A microbiological study of dental calculus. II. The in vitro calcification of microorganisms from dental calculus.},
journal = {Journal of periodontal research},
volume = {13},
number = {4},
pages = {360-366},
doi = {10.1111/j.1600-0765.1978.tb00190.x},
pmid = {149855},
issn = {0022-3484},
mesh = {Bacteria/*metabolism ; Bacteriological Techniques ; Calcium/*metabolism ; Culture Media ; Dental Calculus/*microbiology ; Humans ; },
}
MeSH Terms:
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Bacteria/*metabolism
Bacteriological Techniques
Calcium/*metabolism
Culture Media
Dental Calculus/*microbiology
Humans
RevDate: 2013-11-21
CmpDate: 1975-06-13
A simple test for the diagnosis of absorptive, resorptive and renal hypercalciurias.
The New England journal of medicine, 292(10):497-500.
A test was developed to diagnose various forms of hypercalciuria. A two-hour urine sample after an overnight fast and a four-hour urine sample after 1 g of calcium by mouth were tested for calcium, cyclic AMP and creatinine. The 24 patients with absorptive hypercalciuria had normocalcemia and normal fasting urinary calcium (less than 0.11 mg per milligram of urinary creatnine). Urinary calcium was high (greater than or equal to 0.2 mg per milligram of creatinine) after a calcium load. Of the 28 patients with primary hyperparathyroidism (resorptive hypercalciuria), 25 had hypercalcemia and 21 had high fasting urinary calcium. Urinary cyclic AMP, elevated in 30 per cent of fasting patients, was high (greater than 4.60 mu moles per gram of creatinine) in 82 per cent of cases after calcium load. Six patients with renal hypercalciuria had normocalcemia, high fasting urinary calcium, and high (greater than 6.86 mu moles per gram of creatinine) or high-normal fasting urinary cyclic AMP was normal. This simple test should facilitate the differentiation of various causes of hypercalciuria.
Additional Links: PMID-163960
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PubMed:
Citation:
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@article {pmid163960,
year = {1975},
author = {Pak, CY and Kaplan, R and Bone, H and Townsend, J and Waters, O},
title = {A simple test for the diagnosis of absorptive, resorptive and renal hypercalciurias.},
journal = {The New England journal of medicine},
volume = {292},
number = {10},
pages = {497-500},
doi = {10.1056/NEJM197503062921002},
pmid = {163960},
issn = {0028-4793},
mesh = {Bone Resorption ; Calcium/metabolism/*urine ; Calcium Metabolism Disorders/complications/*diagnosis ; Creatinine/urine ; Cyclic AMP/urine ; Diagnosis, Differential ; Humans ; Hyperparathyroidism/*diagnosis ; *Intestinal Absorption ; Kidney Calculi/*etiology ; Kidney Diseases/*diagnosis ; Kidney Tubules/*physiopathology ; Methods ; },
abstract = {A test was developed to diagnose various forms of hypercalciuria. A two-hour urine sample after an overnight fast and a four-hour urine sample after 1 g of calcium by mouth were tested for calcium, cyclic AMP and creatinine. The 24 patients with absorptive hypercalciuria had normocalcemia and normal fasting urinary calcium (less than 0.11 mg per milligram of urinary creatnine). Urinary calcium was high (greater than or equal to 0.2 mg per milligram of creatinine) after a calcium load. Of the 28 patients with primary hyperparathyroidism (resorptive hypercalciuria), 25 had hypercalcemia and 21 had high fasting urinary calcium. Urinary cyclic AMP, elevated in 30 per cent of fasting patients, was high (greater than 4.60 mu moles per gram of creatinine) in 82 per cent of cases after calcium load. Six patients with renal hypercalciuria had normocalcemia, high fasting urinary calcium, and high (greater than 6.86 mu moles per gram of creatinine) or high-normal fasting urinary cyclic AMP was normal. This simple test should facilitate the differentiation of various causes of hypercalciuria.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Bone Resorption
Calcium/metabolism/*urine
Calcium Metabolism Disorders/complications/*diagnosis
Creatinine/urine
Cyclic AMP/urine
Diagnosis, Differential
Humans
Hyperparathyroidism/*diagnosis
*Intestinal Absorption
Kidney Calculi/*etiology
Kidney Diseases/*diagnosis
Kidney Tubules/*physiopathology
Methods
RevDate: 2013-11-21
CmpDate: 1975-08-18
Recent advances in calcium and phosphorus metabolism.
Annals of clinical and laboratory science, 5(3):176-184.
This review considers the most recent developments concerning the metabolism and homeostasis of calcium and phosphorus. The kinetics of the distribution of calcium, theories of calculus formation, hypercalcemia and hypocalcemia are discussed, as well as the role of parathyroid hormone, thyrocalcitonin and 1,25 dihydroxy Vitamin D(3) in maintaining calcium levels and skeletal integrity. In addition, the role of calcium in enzyme activation and inhibition, muscle and nerve function, and intracellular metabolism are considered.
Additional Links: PMID-166600
PubMed:
Citation:
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@article {pmid166600,
year = {1975},
author = {Schwartz, MK},
title = {Recent advances in calcium and phosphorus metabolism.},
journal = {Annals of clinical and laboratory science},
volume = {5},
number = {3},
pages = {176-184},
pmid = {166600},
issn = {0091-7370},
mesh = {Alkaline Phosphatase/physiology ; Animals ; Bone and Bones/physiology ; Calcification, Physiologic ; Calcitonin/physiology ; Calcium/*metabolism/physiology/urine ; Calcium Radioisotopes ; Calculi/physiopathology ; Cholecalciferol/physiology ; Humans ; Hypercalcemia/physiopathology ; Hypocalcemia/physiopathology ; Intestine, Small/metabolism ; Mitosis ; Muscles/physiology ; Neurons/physiology ; Parathyroid Hormone/physiology ; Phosphorus/*metabolism ; Prostaglandins/physiology ; Rats ; },
abstract = {This review considers the most recent developments concerning the metabolism and homeostasis of calcium and phosphorus. The kinetics of the distribution of calcium, theories of calculus formation, hypercalcemia and hypocalcemia are discussed, as well as the role of parathyroid hormone, thyrocalcitonin and 1,25 dihydroxy Vitamin D(3) in maintaining calcium levels and skeletal integrity. In addition, the role of calcium in enzyme activation and inhibition, muscle and nerve function, and intracellular metabolism are considered.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Alkaline Phosphatase/physiology
Animals
Bone and Bones/physiology
Calcification, Physiologic
Calcitonin/physiology
Calcium/*metabolism/physiology/urine
Calcium Radioisotopes
Calculi/physiopathology
Cholecalciferol/physiology
Humans
Hypercalcemia/physiopathology
Hypocalcemia/physiopathology
Intestine, Small/metabolism
Mitosis
Muscles/physiology
Neurons/physiology
Parathyroid Hormone/physiology
Phosphorus/*metabolism
Prostaglandins/physiology
Rats
RevDate: 2019-05-16
CmpDate: 1975-09-18
Pyrophosphate and diphosphonates in skeletal metabolism. Physiological, clinical and therapeutic aspects.
Clinical orthopaedics and related research.
Pyrophosphate and diphosphonates produce striking results on calcium metabolism in experimental animals and man. Compounds containing P-O-P- bonds (e.g. inorganic pyrophosphate [PP-ii1 or P-C-P bonds (diphosponates) inhibit both the formation and dissolution of calcium phosphate crystals in vitro. PP-i may have a physiological function in regulating calcification and bone turnover, and obnormalities in its metabolism may occur in some human diseases notably hypophosphatasia and pseudogout. Diphosphonates inhibit ectopic calcification, and slow down resorption and bone turnover in several experimental systems in vivo. They have helped in studies of various aspects of the regulation of calcium metabolism. The diphosphonate, disodium ethane-1-hydroxy-1,1-diphosphonate (EHDP) has been shown in clinical studies to be effective against ectopic calcification particularly in myositis ossificans progressiva and in disorders of increased bone resorption such as Paget's diseases and some types of osteoporosis. -99mTechnetium complexes of EHDP, PP-i and other polyphosphates have also recently been used successfully as bone scanning agents.
Additional Links: PMID-166785
Publisher:
PubMed:
Citation:
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@article {pmid166785,
year = {1975},
author = {Russell, RG and Fleisch, H},
title = {Pyrophosphate and diphosphonates in skeletal metabolism. Physiological, clinical and therapeutic aspects.},
journal = {Clinical orthopaedics and related research},
volume = {},
number = {108},
pages = {241-263},
doi = {10.1097/00003086-197505000-00038},
pmid = {166785},
issn = {0009-921X},
mesh = {Animals ; Bone Resorption/drug effects ; Bone and Bones/drug effects/*metabolism ; Calcification, Physiologic/drug effects ; Calcinosis/prevention & control ; Calcium/*metabolism ; Calcium Metabolism Disorders/blood ; Cartilage/drug effects ; Chemical Phenomena ; Chemistry ; Culture Techniques ; Depression, Chemical ; Diphosphates/metabolism/*pharmacology/physiology ; Etidronic Acid/*pharmacology ; Humans ; Hydroxyapatites/metabolism ; Kidney Diseases/prevention & control ; Methylene Chloride/analogs & derivatives/pharmacology ; Organophosphonates/metabolism/*pharmacology/therapeutic use ; Organophosphorus Compounds/*pharmacology ; Osteoporosis/prevention & control ; Urinary Bladder Calculi/prevention & control ; Vitamin D/metabolism ; },
abstract = {Pyrophosphate and diphosphonates produce striking results on calcium metabolism in experimental animals and man. Compounds containing P-O-P- bonds (e.g. inorganic pyrophosphate [PP-ii1 or P-C-P bonds (diphosponates) inhibit both the formation and dissolution of calcium phosphate crystals in vitro. PP-i may have a physiological function in regulating calcification and bone turnover, and obnormalities in its metabolism may occur in some human diseases notably hypophosphatasia and pseudogout. Diphosphonates inhibit ectopic calcification, and slow down resorption and bone turnover in several experimental systems in vivo. They have helped in studies of various aspects of the regulation of calcium metabolism. The diphosphonate, disodium ethane-1-hydroxy-1,1-diphosphonate (EHDP) has been shown in clinical studies to be effective against ectopic calcification particularly in myositis ossificans progressiva and in disorders of increased bone resorption such as Paget's diseases and some types of osteoporosis. -99mTechnetium complexes of EHDP, PP-i and other polyphosphates have also recently been used successfully as bone scanning agents.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Animals
Bone Resorption/drug effects
Bone and Bones/drug effects/*metabolism
Calcification, Physiologic/drug effects
Calcinosis/prevention & control
Calcium/*metabolism
Calcium Metabolism Disorders/blood
Cartilage/drug effects
Chemical Phenomena
Chemistry
Culture Techniques
Depression, Chemical
Diphosphates/metabolism/*pharmacology/physiology
Etidronic Acid/*pharmacology
Humans
Hydroxyapatites/metabolism
Kidney Diseases/prevention & control
Methylene Chloride/analogs & derivatives/pharmacology
Organophosphonates/metabolism/*pharmacology/therapeutic use
Organophosphorus Compounds/*pharmacology
Osteoporosis/prevention & control
Urinary Bladder Calculi/prevention & control
Vitamin D/metabolism
RevDate: 2018-11-13
CmpDate: 1977-06-11
The role of 1 alpha, 25-dihydroxyvitamin D in the mediation of intestinal hyperabsorption of calcium in primary hyperparathyroidism and absorptive hypercalciuria.
The Journal of clinical investigation, 59(5):756-760.
The cuase for the intestinal hyperabsorptionof calcium (Ca) in various forms of hypercalciurias was explored by a careful measurement of plasma 1 alpha, 25-dihydroxycholecalciferol [1 alpha, 25-(OH)I D] and by an assessment of intestinal Ca absorption and of parathyroid function. In 18 cases of primary hyperparathyroidism (PHPT), the mean plasma concentration of 1 alpha, 25-(OH)2D was significantly increased (4.9 +/- 2.2 SD ng/dl vs. 3.4 +/- 0.9 ng/dl for the control group), and was significantly correlated with fractional Ca absorption (alpha) (r = 0.80, P less than 0.001). Plasma 1 alpha, 25-(OH)2D was also correlated with urinary Ca (P less than 0.05), but not with serum Ca or phosphorus (P), P clearance, urinary cyclic AMP, or serum immunoreactive parathyroid hormone. In 21 cases of absorptive hypercalciuria (AH), plasma 1 alpha, 25-(OH)2D was elevated in one-third of cases, and the mean value of 4.5 +/- 1.1 ng/dl was significantly higher than that of the control group (P less than 0.01). Since relative hypoparathyroidism may be present, the normal absolute value of plasma 1 alpha, 25-(OH)2D, found in two-thirds of cases of AH, may be considered to be inappropriately high. Moreover, in the majority of cases of AH, the data points relating plasma 1 alpha, 25-(OH)2D and alpha fell within 95% confidence limits of values found in non-AH groups (including PHPT). The results suggest that the intestinal hyperabsorption of Ca in PHPT aw AH may be vitamin D dependent. However, the disturbance in vitamin D metabolism may not be the sole cause for the high Ca absorption in AH, since in some patients with AH, the intestinal Ca absorption appears to be inapp
Additional Links: PMID-192763
PubMed:
Citation:
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@article {pmid192763,
year = {1977},
author = {Kaplan, RA and Haussler, MR and Deftos, LJ and Bone, H and Pak, CY},
title = {The role of 1 alpha, 25-dihydroxyvitamin D in the mediation of intestinal hyperabsorption of calcium in primary hyperparathyroidism and absorptive hypercalciuria.},
journal = {The Journal of clinical investigation},
volume = {59},
number = {5},
pages = {756-760},
pmid = {192763},
issn = {0021-9738},
mesh = {Adult ; Calcium/blood/metabolism/*urine ; Calcium Metabolism Disorders/*blood ; Cyclic AMP/urine ; Dihydroxycholecalciferols/*blood ; Female ; Humans ; Hydroxycholecalciferols/*blood ; Hyperparathyroidism/*blood/urine ; Intestinal Absorption ; Kidney Calculi/etiology ; Male ; Middle Aged ; Parathyroid Hormone/blood/immunology ; Phosphorus/blood/urine ; },
abstract = {The cuase for the intestinal hyperabsorptionof calcium (Ca) in various forms of hypercalciurias was explored by a careful measurement of plasma 1 alpha, 25-dihydroxycholecalciferol [1 alpha, 25-(OH)I D] and by an assessment of intestinal Ca absorption and of parathyroid function. In 18 cases of primary hyperparathyroidism (PHPT), the mean plasma concentration of 1 alpha, 25-(OH)2D was significantly increased (4.9 +/- 2.2 SD ng/dl vs. 3.4 +/- 0.9 ng/dl for the control group), and was significantly correlated with fractional Ca absorption (alpha) (r = 0.80, P less than 0.001). Plasma 1 alpha, 25-(OH)2D was also correlated with urinary Ca (P less than 0.05), but not with serum Ca or phosphorus (P), P clearance, urinary cyclic AMP, or serum immunoreactive parathyroid hormone. In 21 cases of absorptive hypercalciuria (AH), plasma 1 alpha, 25-(OH)2D was elevated in one-third of cases, and the mean value of 4.5 +/- 1.1 ng/dl was significantly higher than that of the control group (P less than 0.01). Since relative hypoparathyroidism may be present, the normal absolute value of plasma 1 alpha, 25-(OH)2D, found in two-thirds of cases of AH, may be considered to be inappropriately high. Moreover, in the majority of cases of AH, the data points relating plasma 1 alpha, 25-(OH)2D and alpha fell within 95% confidence limits of values found in non-AH groups (including PHPT). The results suggest that the intestinal hyperabsorption of Ca in PHPT aw AH may be vitamin D dependent. However, the disturbance in vitamin D metabolism may not be the sole cause for the high Ca absorption in AH, since in some patients with AH, the intestinal Ca absorption appears to be inapp},
}
MeSH Terms:
show MeSH Terms
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Adult
Calcium/blood/metabolism/*urine
Calcium Metabolism Disorders/*blood
Cyclic AMP/urine
Dihydroxycholecalciferols/*blood
Female
Humans
Hydroxycholecalciferols/*blood
Hyperparathyroidism/*blood/urine
Intestinal Absorption
Kidney Calculi/etiology
Male
Middle Aged
Parathyroid Hormone/blood/immunology
Phosphorus/blood/urine
RevDate: 2019-06-22
CmpDate: 1977-10-14
Diphosphonates. Mode of action and clinical applications.
Advances in experimental medicine and biology, 81:279-289.
Additional Links: PMID-197817
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PubMed:
Citation:
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@article {pmid197817,
year = {1977},
author = {Fleisch, H and Fast, D and Rizzoli, R and Trechsel, U and Bonjour, JP},
title = {Diphosphonates. Mode of action and clinical applications.},
journal = {Advances in experimental medicine and biology},
volume = {81},
number = {},
pages = {279-289},
doi = {10.1007/978-1-4613-4217-5_27},
pmid = {197817},
issn = {0065-2598},
mesh = {Bone Resorption/drug effects ; Calcification, Physiologic/drug effects ; Calcinosis/drug therapy ; Calcium/*metabolism ; Calcium Phosphates ; Chemical Precipitation ; Dental Calculus/prevention & control ; Dihydroxycholecalciferols/metabolism ; Diphosphates/metabolism ; *Diphosphonates/pharmacology/therapeutic use ; Humans ; Osteitis Deformans/drug therapy ; Osteoporosis/drug therapy ; Phosphates/*metabolism ; Phosphoric Monoester Hydrolases/antagonists & inhibitors ; Radionuclide Imaging/methods ; Structure-Activity Relationship ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Bone Resorption/drug effects
Calcification, Physiologic/drug effects
Calcinosis/drug therapy
Calcium/*metabolism
Calcium Phosphates
Chemical Precipitation
Dental Calculus/prevention & control
Dihydroxycholecalciferols/metabolism
Diphosphates/metabolism
*Diphosphonates/pharmacology/therapeutic use
Humans
Osteitis Deformans/drug therapy
Osteoporosis/drug therapy
Phosphates/*metabolism
Phosphoric Monoester Hydrolases/antagonists & inhibitors
Radionuclide Imaging/methods
Structure-Activity Relationship
RevDate: 2013-11-21
CmpDate: 1977-11-25
[Effect of orthophosphate, pyrophosphate and diphosphonate in urinary-calculi metaphylaxis].
Zeitschrift fur Urologie und Nephrologie, 70(6):449-452.
Additional Links: PMID-198996
PubMed:
Citation:
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@article {pmid198996,
year = {1977},
author = {Baumann, JM},
title = {[Effect of orthophosphate, pyrophosphate and diphosphonate in urinary-calculi metaphylaxis].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {70},
number = {6},
pages = {449-452},
pmid = {198996},
issn = {0044-3611},
mesh = {Calcium/metabolism ; Diphosphates/adverse effects/*therapeutic use ; Diphosphonates/adverse effects/*therapeutic use ; Humans ; Kidney Calculi/*drug therapy/etiology/metabolism ; Muscular Diseases/chemically induced ; Pain/chemically induced ; Phosphates/adverse effects/*therapeutic use ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/metabolism
Diphosphates/adverse effects/*therapeutic use
Diphosphonates/adverse effects/*therapeutic use
Humans
Kidney Calculi/*drug therapy/etiology/metabolism
Muscular Diseases/chemically induced
Pain/chemically induced
Phosphates/adverse effects/*therapeutic use
RevDate: 2013-11-21
CmpDate: 1978-04-17
Cyclic AMP urinary excretion and radiocalcium intestinal absorption in hypercalciurias.
Bollettino della Societa italiana di biologia sperimentale, 53(14):1178-1182.
Additional Links: PMID-203297
PubMed:
Citation:
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@article {pmid203297,
year = {1977},
author = {Vattimo, GC and Galli, M and Nuti, R and Loré, F and Nardi, P and Orlandini, G},
title = {Cyclic AMP urinary excretion and radiocalcium intestinal absorption in hypercalciurias.},
journal = {Bollettino della Societa italiana di biologia sperimentale},
volume = {53},
number = {14},
pages = {1178-1182},
pmid = {203297},
issn = {0037-8771},
mesh = {Calcium/urine ; Calcium Metabolism Disorders/*metabolism ; Cyclic AMP/*urine ; Humans ; Intestinal Absorption ; Urinary Calculi/metabolism ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/urine
Calcium Metabolism Disorders/*metabolism
Cyclic AMP/*urine
Humans
Intestinal Absorption
Urinary Calculi/metabolism
RevDate: 2015-05-23
CmpDate: 1978-07-24
[Etiology of hypercalciuria in recurrent kidney calculi].
Lakartidningen, 75(19):1910-1911.
Additional Links: PMID-206792
PubMed:
Citation:
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@article {pmid206792,
year = {1978},
author = {Backman, U and Danielson, BG and Johansson, G and Jung, B and Ljunghall, S and Wibell, L and Walinder, O},
title = {[Etiology of hypercalciuria in recurrent kidney calculi].},
journal = {Lakartidningen},
volume = {75},
number = {19},
pages = {1910-1911},
pmid = {206792},
issn = {0023-7205},
mesh = {Calcium/*urine ; Calcium Metabolism Disorders/*etiology ; Cyclic AMP/urine ; Digestive System/metabolism ; Female ; Humans ; Kidney Calculi/*metabolism ; Male ; Recurrence ; Risk ; },
}
MeSH Terms:
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Calcium/*urine
Calcium Metabolism Disorders/*etiology
Cyclic AMP/urine
Digestive System/metabolism
Female
Humans
Kidney Calculi/*metabolism
Male
Recurrence
Risk
RevDate: 2013-11-21
CmpDate: 1978-12-02
[Therapeutic measures in recurrent calcium oxalate nephrolithiasis].
Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis, 67(38):1385-1388.
Additional Links: PMID-211499
PubMed:
Citation:
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@article {pmid211499,
year = {1978},
author = {Tschöpe, W and Schmidt-Gayk, H and Ritz, E},
title = {[Therapeutic measures in recurrent calcium oxalate nephrolithiasis].},
journal = {Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis},
volume = {67},
number = {38},
pages = {1385-1388},
pmid = {211499},
issn = {1013-2058},
mesh = {Benzothiadiazines ; Calcium/*metabolism ; Cellulose/therapeutic use ; Diet Therapy ; Diphosphates/metabolism ; Diuretics ; Humans ; Intestinal Absorption/drug effects ; Ion Exchange ; Phosphates/therapeutic use ; Recurrence ; Sodium/therapeutic use ; Sodium Chloride Symporter Inhibitors/therapeutic use ; Urinary Calculi/*therapy ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Benzothiadiazines
Calcium/*metabolism
Cellulose/therapeutic use
Diet Therapy
Diphosphates/metabolism
Diuretics
Humans
Intestinal Absorption/drug effects
Ion Exchange
Phosphates/therapeutic use
Recurrence
Sodium/therapeutic use
Sodium Chloride Symporter Inhibitors/therapeutic use
Urinary Calculi/*therapy
RevDate: 2013-11-21
CmpDate: 1979-09-01
[Kidney and parathyroid hormone].
Minerva medica, 70(27):1951-1955.
Additional Links: PMID-221862
PubMed:
Citation:
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@article {pmid221862,
year = {1979},
author = {Bertolissi, F and Maschio, M and Benedetti, A},
title = {[Kidney and parathyroid hormone].},
journal = {Minerva medica},
volume = {70},
number = {27},
pages = {1951-1955},
pmid = {221862},
issn = {0026-4806},
mesh = {Calcium/metabolism ; Cyclic AMP/metabolism ; Humans ; Hyperparathyroidism/metabolism ; Hypoparathyroidism/metabolism ; Kidney/metabolism/*physiology ; Kidney Calculi/metabolism ; Kidney Failure, Chronic/metabolism ; Kidney Tubules/physiology ; Parathyroid Hormone/*physiology ; Phosphorus/urine ; },
}
MeSH Terms:
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Calcium/metabolism
Cyclic AMP/metabolism
Humans
Hyperparathyroidism/metabolism
Hypoparathyroidism/metabolism
Kidney/metabolism/*physiology
Kidney Calculi/metabolism
Kidney Failure, Chronic/metabolism
Kidney Tubules/physiology
Parathyroid Hormone/*physiology
Phosphorus/urine
RevDate: 2013-11-21
CmpDate: 1981-09-15
[Hyperparathyroidism and recurrent calcic nephrolithiasis].
Minerva nefrologica, 26(3):305-308.
Additional Links: PMID-262269
PubMed:
Citation:
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@article {pmid262269,
year = {1979},
author = {Graziani, G and Mioni, G and Aroldi, A and Surian, M and Cecchettin, M and Galmozzi, C and Lorenzano, E and Bonforte, G},
title = {[Hyperparathyroidism and recurrent calcic nephrolithiasis].},
journal = {Minerva nefrologica},
volume = {26},
number = {3},
pages = {305-308},
pmid = {262269},
issn = {0026-4873},
mesh = {Calcium/metabolism/urine ; Humans ; Hyperparathyroidism/*complications/metabolism ; Kidney Calculi/*complications/metabolism ; Phosphorus/metabolism ; },
}
MeSH Terms:
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Calcium/metabolism/urine
Humans
Hyperparathyroidism/*complications/metabolism
Kidney Calculi/*complications/metabolism
Phosphorus/metabolism
RevDate: 2013-11-21
CmpDate: 1981-09-15
[Incidence and genesis of idiopathic hypercalciuria in calcic urolithiasis in a southern area].
Minerva nefrologica, 26(3):321-323.
Additional Links: PMID-262272
PubMed:
Citation:
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@article {pmid262272,
year = {1979},
author = {Ciccarelli, C and Pizzarelli, F and Parlongo, S and Maggiore, Q},
title = {[Incidence and genesis of idiopathic hypercalciuria in calcic urolithiasis in a southern area].},
journal = {Minerva nefrologica},
volume = {26},
number = {3},
pages = {321-323},
pmid = {262272},
issn = {0026-4873},
mesh = {Calcium/*urine ; Calcium Metabolism Disorders/*complications ; Female ; Humans ; Hyperparathyroidism/*complications ; Male ; Phosphates/metabolism ; Uric Acid/metabolism ; Urinary Calculi/*complications ; },
}
MeSH Terms:
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Calcium/*urine
Calcium Metabolism Disorders/*complications
Female
Humans
Hyperparathyroidism/*complications
Male
Phosphates/metabolism
Uric Acid/metabolism
Urinary Calculi/*complications
RevDate: 2017-02-14
CmpDate: 1977-04-30
Proteolipid and calculus matrix calcification in vitro.
Journal of dental research, 56(2):140-142.
The initiator of calculus matrix calcification, in vitro, was isolated. Crude phospholipid, known to contain the factor, was separated into five fractions by column chromatography. A single protein-containing fraction induced apatite formation during incubation. The nucleating fraction was indentified as a proteolipid.
Additional Links: PMID-264880
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PubMed:
Citation:
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@article {pmid264880,
year = {1977},
author = {Ennever, J and Vocel, JJ and Riggan, LJ and Paloski, SB},
title = {Proteolipid and calculus matrix calcification in vitro.},
journal = {Journal of dental research},
volume = {56},
number = {2},
pages = {140-142},
doi = {10.1177/00220345770560020701},
pmid = {264880},
issn = {0022-0345},
mesh = {Actinomycetaceae/physiology ; Calcium/*metabolism ; Dental Calculus/*metabolism ; History, 18th Century ; Phospholipids/physiology ; Proteolipids/*physiology ; },
abstract = {The initiator of calculus matrix calcification, in vitro, was isolated. Crude phospholipid, known to contain the factor, was separated into five fractions by column chromatography. A single protein-containing fraction induced apatite formation during incubation. The nucleating fraction was indentified as a proteolipid.},
}
MeSH Terms:
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hide MeSH Terms
Actinomycetaceae/physiology
Calcium/*metabolism
Dental Calculus/*metabolism
History, 18th Century
Phospholipids/physiology
Proteolipids/*physiology
RevDate: 2018-02-16
CmpDate: 1977-05-12
Caries reduction in rats by phosphate, magnesium and fluoride additions to diet with modifications of dental calculus and calcium of the kidneys and aorta.
Caries research, 11(2):100-108.
Additional Links: PMID-265186
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PubMed:
Citation:
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@article {pmid265186,
year = {1977},
author = {Luoma, H and Nuuja, T},
title = {Caries reduction in rats by phosphate, magnesium and fluoride additions to diet with modifications of dental calculus and calcium of the kidneys and aorta.},
journal = {Caries research},
volume = {11},
number = {2},
pages = {100-108},
doi = {10.1159/000260256},
pmid = {265186},
issn = {0008-6568},
mesh = {Animals ; Aorta/metabolism ; Calcium/metabolism ; Calculi/etiology ; Dental Calculus/etiology ; Dental Caries/*prevention & control ; *Diet ; Female ; Fluorides/*pharmacology ; Growth/drug effects ; Kidney/metabolism ; Kidney Calculi/etiology ; Magnesium/*pharmacology ; Magnesium Deficiency ; Male ; Phosphates/*pharmacology ; Rats ; Vascular Diseases/etiology ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Animals
Aorta/metabolism
Calcium/metabolism
Calculi/etiology
Dental Calculus/etiology
Dental Caries/*prevention & control
*Diet
Female
Fluorides/*pharmacology
Growth/drug effects
Kidney/metabolism
Kidney Calculi/etiology
Magnesium/*pharmacology
Magnesium Deficiency
Male
Phosphates/*pharmacology
Rats
Vascular Diseases/etiology
RevDate: 2013-11-21
CmpDate: 1978-02-18
Renal calcium and its implications.
The West Virginia medical journal, 73(12):319-321.
Additional Links: PMID-271421
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Citation:
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@article {pmid271421,
year = {1977},
author = {Pfister, AK},
title = {Renal calcium and its implications.},
journal = {The West Virginia medical journal},
volume = {73},
number = {12},
pages = {319-321},
pmid = {271421},
issn = {0043-3284},
mesh = {Calcium/*metabolism/urine ; Humans ; Kidney Calculi/etiology/*metabolism/therapy ; },
}
MeSH Terms:
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Calcium/*metabolism/urine
Humans
Kidney Calculi/etiology/*metabolism/therapy
RevDate: 2004-11-17
CmpDate: 1978-12-27
[Diphosphonates and calcified tissues].
L' Information dentaire, 59(41):21-31.
Additional Links: PMID-279501
PubMed:
Citation:
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@article {pmid279501,
year = {1977},
author = {Clergeau-Guerithault, S},
title = {[Diphosphonates and calcified tissues].},
journal = {L' Information dentaire},
volume = {59},
number = {41},
pages = {21-31},
pmid = {279501},
issn = {0020-0018},
mesh = {Calcium Metabolism Disorders/*drug therapy ; Dental Calculus/prevention & control ; Dental Caries/prevention & control ; Diphosphonates/*therapeutic use ; Humans ; },
}
MeSH Terms:
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hide MeSH Terms
Calcium Metabolism Disorders/*drug therapy
Dental Calculus/prevention & control
Dental Caries/prevention & control
Diphosphonates/*therapeutic use
Humans
RevDate: 2019-12-10
CmpDate: 1977-10-31
[Results of clinical trials with the cation exchange preparation campanyl in the therapy and metaphylaxis of calcium-containing urinary calculi (author's transl)].
Der Urologe. Ausg. A, 16(4):197-203.
The efficacy of the cation exchange preparation Campanyl (T1286) was tested in the treatment and metaphylaxis of calcium-containing urinary calculi. For this purpose, in vitro experiments, animal exeriments, orienting clinical studies in 79 patients, and a long term clinical trial in 42 patients over 12 months were undertaken; 22 of the latter patients are also still being treated with the cation exchange preparation, the observation period for 15 patients being 3 1/2 years and 2 1/2 years for 7 patients. As a result of these studies a lowering of the medium calcium excretion and a reduction of calculus discharge by more than half was achieved in the patients being treated with the cation exchanger, without restriction of calcium in the diet. There was not yet seen litholysis. Serious side effects or an influence on the serum electrolytes was not recorded with Campanyl (T1286).
Additional Links: PMID-331626
PubMed:
Citation:
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@article {pmid331626,
year = {1977},
author = {Rugendorff, EW and Schneider, HJ and Gundlach, G and Behrendt, WA and Kuhn, D and Gundlach, D},
title = {[Results of clinical trials with the cation exchange preparation campanyl in the therapy and metaphylaxis of calcium-containing urinary calculi (author's transl)].},
journal = {Der Urologe. Ausg. A},
volume = {16},
number = {4},
pages = {197-203},
pmid = {331626},
issn = {0340-2592},
mesh = {Calcium/*metabolism ; Carboxymethylcellulose Sodium/therapeutic use ; Cation Exchange Resins/*therapeutic use ; Clinical Trials as Topic ; Drug Combinations ; Drug Evaluation ; Female ; Humans ; Ion Exchange Resins/*therapeutic use ; Male ; Polyvinyls/*therapeutic use ; Sorbitol/therapeutic use ; Urinary Calculi/*drug therapy/metabolism ; },
abstract = {The efficacy of the cation exchange preparation Campanyl (T1286) was tested in the treatment and metaphylaxis of calcium-containing urinary calculi. For this purpose, in vitro experiments, animal exeriments, orienting clinical studies in 79 patients, and a long term clinical trial in 42 patients over 12 months were undertaken; 22 of the latter patients are also still being treated with the cation exchange preparation, the observation period for 15 patients being 3 1/2 years and 2 1/2 years for 7 patients. As a result of these studies a lowering of the medium calcium excretion and a reduction of calculus discharge by more than half was achieved in the patients being treated with the cation exchanger, without restriction of calcium in the diet. There was not yet seen litholysis. Serious side effects or an influence on the serum electrolytes was not recorded with Campanyl (T1286).},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/*metabolism
Carboxymethylcellulose Sodium/therapeutic use
Cation Exchange Resins/*therapeutic use
Clinical Trials as Topic
Drug Combinations
Drug Evaluation
Female
Humans
Ion Exchange Resins/*therapeutic use
Male
Polyvinyls/*therapeutic use
Sorbitol/therapeutic use
Urinary Calculi/*drug therapy/metabolism
RevDate: 2013-11-21
CmpDate: 1977-11-25
[Experiences with nephrolith in the treatment of patients with kidney calculi].
Zeitschrift fur Urologie und Nephrologie, 70(6):429-435.
In patients with urolithiasis "Nephrolith" has a favourable influence on many parameters which are responsible for the development of renal calculi. I could never observe a complete dissolution of the urinary calculi.
Additional Links: PMID-333815
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Citation:
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@article {pmid333815,
year = {1977},
author = {Modliński, L},
title = {[Experiences with nephrolith in the treatment of patients with kidney calculi].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {70},
number = {6},
pages = {429-435},
pmid = {333815},
issn = {0044-3611},
mesh = {Adolescent ; Adult ; Aged ; Bacterial Infections/etiology ; Calcium/metabolism ; Child ; Child, Preschool ; Clinical Trials as Topic ; Drug Combinations ; Drug Evaluation ; Female ; Humans ; Khellin/*therapeutic use ; Kidney Calculi/complications/*drug therapy/metabolism ; Male ; Middle Aged ; Oxalates/metabolism ; Phosphates/metabolism ; Sulfonamides/*therapeutic use ; Urinary Calculi/drug therapy ; },
abstract = {In patients with urolithiasis "Nephrolith" has a favourable influence on many parameters which are responsible for the development of renal calculi. I could never observe a complete dissolution of the urinary calculi.},
}
MeSH Terms:
show MeSH Terms
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Adolescent
Adult
Aged
Bacterial Infections/etiology
Calcium/metabolism
Child
Child, Preschool
Clinical Trials as Topic
Drug Combinations
Drug Evaluation
Female
Humans
Khellin/*therapeutic use
Kidney Calculi/complications/*drug therapy/metabolism
Male
Middle Aged
Oxalates/metabolism
Phosphates/metabolism
Sulfonamides/*therapeutic use
Urinary Calculi/drug therapy
RevDate: 2013-11-21
CmpDate: 1978-02-18
Nutrition and urolithiasis.
The New England journal of medicine, 298(2):87-89.
Additional Links: PMID-339082
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PubMed:
Citation:
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@article {pmid339082,
year = {1978},
author = {Smith, LH and Van Den Berg, CJ and Wilson, DM},
title = {Nutrition and urolithiasis.},
journal = {The New England journal of medicine},
volume = {298},
number = {2},
pages = {87-89},
doi = {10.1056/NEJM197801122980207},
pmid = {339082},
issn = {0028-4793},
mesh = {Calcium/metabolism ; Cystine/metabolism ; Cystinuria/metabolism ; Humans ; *Nutritional Physiological Phenomena ; Oxalates/metabolism ; Phosphates/metabolism ; Uric Acid/metabolism ; Urinary Calculi/diet therapy/*metabolism/prevention & control ; },
}
MeSH Terms:
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hide MeSH Terms
Calcium/metabolism
Cystine/metabolism
Cystinuria/metabolism
Humans
*Nutritional Physiological Phenomena
Oxalates/metabolism
Phosphates/metabolism
Uric Acid/metabolism
Urinary Calculi/diet therapy/*metabolism/prevention & control
RevDate: 2019-08-13
CmpDate: 1978-04-26
Diagnostic procedures in disorders of calcium metabolism.
Clinical endocrinology, 8(1):55-67.
The investigation of a patient with a suspected disorder of calcium metabolism is most rapidly and efficiently performed by observing a standard procedure, the initial stages of which are simple and mandatory, the later stages more complex and determined by the initial results and the depth of investigation required. Needless to say, the investigations are preceded by history taking and routine physical examination. The following account is a summary of available procedures, most of which have been described elsewhere in greater detail (Nordin et al., 1976a).
Additional Links: PMID-342147
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PubMed:
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@article {pmid342147,
year = {1978},
author = {Nordin, BE},
title = {Diagnostic procedures in disorders of calcium metabolism.},
journal = {Clinical endocrinology},
volume = {8},
number = {1},
pages = {55-67},
doi = {10.1111/j.1365-2265.1978.tb01350.x},
pmid = {342147},
issn = {0300-0664},
mesh = {Absorptiometry, Photon ; Bone Resorption ; Bone and Bones/diagnostic imaging ; Calcium/blood/urine ; Calcium Metabolism Disorders/blood/*diagnosis/urine ; Creatinine/urine ; Glomerular Filtration Rate ; Humans ; Hydroxyproline/urine ; Intestinal Absorption ; Kidney Calculi/diagnosis/urine ; Phosphates/blood/urine ; },
abstract = {The investigation of a patient with a suspected disorder of calcium metabolism is most rapidly and efficiently performed by observing a standard procedure, the initial stages of which are simple and mandatory, the later stages more complex and determined by the initial results and the depth of investigation required. Needless to say, the investigations are preceded by history taking and routine physical examination. The following account is a summary of available procedures, most of which have been described elsewhere in greater detail (Nordin et al., 1976a).},
}
MeSH Terms:
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hide MeSH Terms
Absorptiometry, Photon
Bone Resorption
Bone and Bones/diagnostic imaging
Calcium/blood/urine
Calcium Metabolism Disorders/blood/*diagnosis/urine
Creatinine/urine
Glomerular Filtration Rate
Humans
Hydroxyproline/urine
Intestinal Absorption
Kidney Calculi/diagnosis/urine
Phosphates/blood/urine
RevDate: 2013-11-21
CmpDate: 1979-06-11
[Antirelapse action of the flowers of the blue cornflower in urolithiasis].
Zhurnal eksperimental'noi i klinicheskoi meditsiny, 18(6):110-114.
Additional Links: PMID-373328
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Citation:
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@article {pmid373328,
year = {1978},
author = {Bablumian, IuA},
title = {[Antirelapse action of the flowers of the blue cornflower in urolithiasis].},
journal = {Zhurnal eksperimental'noi i klinicheskoi meditsiny},
volume = {18},
number = {6},
pages = {110-114},
pmid = {373328},
issn = {0514-7484},
mesh = {Adult ; Calcium/metabolism ; Clinical Trials as Topic ; Diuresis/drug effects ; Drug Evaluation ; Female ; Humans ; Male ; Middle Aged ; Phosphorus/metabolism ; *Plants, Medicinal ; Recurrence ; Time Factors ; Uric Acid/metabolism ; Urinary Calculi/*drug therapy/metabolism ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Calcium/metabolism
Clinical Trials as Topic
Diuresis/drug effects
Drug Evaluation
Female
Humans
Male
Middle Aged
Phosphorus/metabolism
*Plants, Medicinal
Recurrence
Time Factors
Uric Acid/metabolism
Urinary Calculi/*drug therapy/metabolism
RevDate: 2019-09-11
CmpDate: 1980-01-24
A survey of calcium urolithiasis in normocalcemic hypercalciuria: possible role of nutrients and diet-mediated factors.
Urological research, 7(3):149-155.
Three types of hypercalciuria are described; their existence and frequent association with calcium urolithiasis in humans are accepted. Various dietary factors such as minerals, electrolytes, fluids, vitamin D, carbohydrates, proteins are discussed with regard to their ability to alter the nature and the degree of calcium excretion following their ingestion. It is emphasised that at present we have only limited knowledge on the chain of events linking calorie intake and the response of the kidney.
Additional Links: PMID-388796
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@article {pmid388796,
year = {1979},
author = {Schwille, PO},
title = {A survey of calcium urolithiasis in normocalcemic hypercalciuria: possible role of nutrients and diet-mediated factors.},
journal = {Urological research},
volume = {7},
number = {3},
pages = {149-155},
pmid = {388796},
issn = {0300-5623},
mesh = {Adult ; Aged ; Animals ; Calcium/blood/*metabolism/urine ; *Diet ; Dietary Carbohydrates/metabolism ; Dietary Fats/metabolism ; Dietary Proteins/metabolism ; Female ; Humans ; Kidney/*metabolism ; Magnesium Deficiency/metabolism ; Male ; Middle Aged ; Oxalates/urine ; Phosphates/metabolism ; Rats ; Sodium/metabolism ; Urinary Calculi/*metabolism ; Vitamin D/metabolism ; },
abstract = {Three types of hypercalciuria are described; their existence and frequent association with calcium urolithiasis in humans are accepted. Various dietary factors such as minerals, electrolytes, fluids, vitamin D, carbohydrates, proteins are discussed with regard to their ability to alter the nature and the degree of calcium excretion following their ingestion. It is emphasised that at present we have only limited knowledge on the chain of events linking calorie intake and the response of the kidney.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Aged
Animals
Calcium/blood/*metabolism/urine
*Diet
Dietary Carbohydrates/metabolism
Dietary Fats/metabolism
Dietary Proteins/metabolism
Female
Humans
Kidney/*metabolism
Magnesium Deficiency/metabolism
Male
Middle Aged
Oxalates/urine
Phosphates/metabolism
Rats
Sodium/metabolism
Urinary Calculi/*metabolism
Vitamin D/metabolism
RevDate: 2013-11-21
CmpDate: 1980-09-28
[What has been achieved in the therapy of calcium nephrolithiasis?].
Der Internist, 20(12):599-606.
Additional Links: PMID-398355
PubMed:
Citation:
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@article {pmid398355,
year = {1979},
author = {Tschöpe, W and Ritz, E},
title = {[What has been achieved in the therapy of calcium nephrolithiasis?].},
journal = {Der Internist},
volume = {20},
number = {12},
pages = {599-606},
pmid = {398355},
issn = {0020-9554},
mesh = {Adult ; Calcium/*metabolism ; Humans ; Kidney Calculi/*drug therapy/metabolism ; Male ; Middle Aged ; Recurrence ; },
}
MeSH Terms:
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hide MeSH Terms
Adult
Calcium/*metabolism
Humans
Kidney Calculi/*drug therapy/metabolism
Male
Middle Aged
Recurrence
RevDate: 2019-06-19
CmpDate: 1979-07-16
Urinary saturation measurements in calcium nephrolithiasis.
Annals of internal medicine, 90(2):180-184.
Urinary saturation with respect to calcium oxalate monohydrate was measured in 111 consecutive patients with calcium nephrolithiasis. Each patient also was evaluated by a detailed conventional metabolic protocol. Patients with idiopathic hypercalciuria produced abnormally oversaturated urine more frequently than normal subjects and normocalciuric patients, but normocalciuric patients had unexpectedly high levels of urine saturation. Measuring levels of calcium concentration, oxalate concentration, or the chemical concentration product of calcium and oxalate in urine did not predict oversaturation. During thiazide treatment, saturation level tended to fall if it was initially elevated, whether the patient was hypercalciuric or not. Patients whose urine was not remarkably oversaturated showed no tendency to elaborate even less saturated urine during thiazide treatment; instead, the average calcium oxalate saturation level remained constant. Direct urine saturation measurements can detect a small but significant number of normocalciuric patients who have marked oversaturation with respect to calcium oxalate and appear to benefit from treatment.
Additional Links: PMID-443649
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@article {pmid443649,
year = {1979},
author = {Weber, DV and Coe, FL and Parks, JH and Dunn, MS and Tembe, V},
title = {Urinary saturation measurements in calcium nephrolithiasis.},
journal = {Annals of internal medicine},
volume = {90},
number = {2},
pages = {180-184},
doi = {10.7326/0003-4819-90-2-180},
pmid = {443649},
issn = {0003-4819},
mesh = {Calcium/metabolism/*urine ; Calcium Oxalate/*urine ; Chlorthalidone/therapeutic use ; Female ; Humans ; Kidney Calculi/drug therapy/metabolism/*urine ; Male ; Time Factors ; Trichlormethiazide/therapeutic use ; },
abstract = {Urinary saturation with respect to calcium oxalate monohydrate was measured in 111 consecutive patients with calcium nephrolithiasis. Each patient also was evaluated by a detailed conventional metabolic protocol. Patients with idiopathic hypercalciuria produced abnormally oversaturated urine more frequently than normal subjects and normocalciuric patients, but normocalciuric patients had unexpectedly high levels of urine saturation. Measuring levels of calcium concentration, oxalate concentration, or the chemical concentration product of calcium and oxalate in urine did not predict oversaturation. During thiazide treatment, saturation level tended to fall if it was initially elevated, whether the patient was hypercalciuric or not. Patients whose urine was not remarkably oversaturated showed no tendency to elaborate even less saturated urine during thiazide treatment; instead, the average calcium oxalate saturation level remained constant. Direct urine saturation measurements can detect a small but significant number of normocalciuric patients who have marked oversaturation with respect to calcium oxalate and appear to benefit from treatment.},
}
MeSH Terms:
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Calcium/metabolism/*urine
Calcium Oxalate/*urine
Chlorthalidone/therapeutic use
Female
Humans
Kidney Calculi/drug therapy/metabolism/*urine
Male
Time Factors
Trichlormethiazide/therapeutic use
RevDate: 2016-11-23
CmpDate: 1979-08-29
Clinical evaluation of renal lithiasis.
Geriatrics, 34(7):35-42.
Determining metabolic activity in urolithiasis may avoid excessive laboratory tests and unnecessary treatment. A simple regimen such as increased oral fluids is often effective therapy in metabolically inactive disease. Most cases of renal lithiasis are idiopathic, but a complete examination and laboratory work-up will usually establish an accurate etiologic diagnosis.
Additional Links: PMID-447082
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@article {pmid447082,
year = {1979},
author = {Van Den Berg, CJ},
title = {Clinical evaluation of renal lithiasis.},
journal = {Geriatrics},
volume = {34},
number = {7},
pages = {35-42},
pmid = {447082},
issn = {0016-867X},
mesh = {Aged ; Calcium/urine ; Calcium Metabolism Disorders/complications/diagnosis ; Cystinuria/complications/diagnosis ; Humans ; Male ; Medical History Taking ; Oxalates/urine ; Physical Examination ; Radiography ; Uric Acid/urine ; Urinary Calculi/diagnostic imaging/*etiology/metabolism ; Xanthines/urine ; },
abstract = {Determining metabolic activity in urolithiasis may avoid excessive laboratory tests and unnecessary treatment. A simple regimen such as increased oral fluids is often effective therapy in metabolically inactive disease. Most cases of renal lithiasis are idiopathic, but a complete examination and laboratory work-up will usually establish an accurate etiologic diagnosis.},
}
MeSH Terms:
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Aged
Calcium/urine
Calcium Metabolism Disorders/complications/diagnosis
Cystinuria/complications/diagnosis
Humans
Male
Medical History Taking
Oxalates/urine
Physical Examination
Radiography
Uric Acid/urine
Urinary Calculi/diagnostic imaging/*etiology/metabolism
Xanthines/urine
RevDate: 2013-11-21
CmpDate: 1979-09-27
Calcium-45 uptake in blood and urine in experimental urolithiasis and idiopathic stoneformers.
The Indian journal of medical research, 69:639-644.
Additional Links: PMID-457206
PubMed:
Citation:
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@article {pmid457206,
year = {1979},
author = {Thind, SK and Nath, R and Aggarwal, SK and Rao, MS and Bapna, BC},
title = {Calcium-45 uptake in blood and urine in experimental urolithiasis and idiopathic stoneformers.},
journal = {The Indian journal of medical research},
volume = {69},
number = {},
pages = {639-644},
pmid = {457206},
issn = {0971-5916},
mesh = {Adult ; Animals ; Calcium/*metabolism ; Humans ; Male ; Rats ; Urinary Calculi/*metabolism ; },
}
MeSH Terms:
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Adult
Animals
Calcium/*metabolism
Humans
Male
Rats
Urinary Calculi/*metabolism
RevDate: 2013-11-21
CmpDate: 1979-10-24
[Urinary oxalate in recurrent calcium urolithiasis with and without hyperfunctioning parathyroid glands and in healthy controls. influence of age and season (author's transl)].
Der Urologe. Ausg. A, 18(4):215-224.
Additional Links: PMID-473457
PubMed:
Citation:
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@article {pmid473457,
year = {1979},
author = {Schwille, PO and Paulus, M and Scholz, D and Sigel, A and Wilhelm, E},
title = {[Urinary oxalate in recurrent calcium urolithiasis with and without hyperfunctioning parathyroid glands and in healthy controls. influence of age and season (author's transl)].},
journal = {Der Urologe. Ausg. A},
volume = {18},
number = {4},
pages = {215-224},
pmid = {473457},
issn = {0340-2592},
mesh = {Adult ; Age Factors ; Body Weight ; Calcium/metabolism ; Creatinine/urine ; Female ; Humans ; Hyperparathyroidism, Secondary/complications/*urine ; Male ; Middle Aged ; Oxalates/*urine ; Recurrence ; Seasons ; Urinary Calculi/complications/metabolism/*urine ; },
}
MeSH Terms:
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hide MeSH Terms
Adult
Age Factors
Body Weight
Calcium/metabolism
Creatinine/urine
Female
Humans
Hyperparathyroidism, Secondary/complications/*urine
Male
Middle Aged
Oxalates/*urine
Recurrence
Seasons
Urinary Calculi/complications/metabolism/*urine
RevDate: 2018-03-30
CmpDate: 1979-11-28
Plasma 1,25-(OH)2-vitamin D concentrations and net intestinal calcium, phosphate, and magnesium absorption in humans.
The American journal of clinical nutrition, 32(10):2052-2060.
We evaluated the relationship between plasma concentrations of the renal hormone 1,25-(OH)2-vitamin D and net intestinal absorption of Ca, PO4, and Mg in vitamin D-replete patients eating similar diets, who had undetectable, normal or elevated plasma 1,25-(OH)2-D levels, Net intestinal Ca absorption was positively correlated to plasma 1,25-(OH)2-D concentrations: percentage dietary Ca absorbed = 10 + 0.17 x plasma total 1,25-(OH)2-3, pmole/liter, r = + 0.58; P less than 0.001. By contrast, there was no significant correlation between PO4 or Mg absorption and plasma 1,25-(OH)2-D concentrations. Moreover, significant quantities of PO4 and Mg were absorbed in the absence of detectable plasma 1,25-(OH)2-D. We conclude that net intestinal Ca absorption is critically dependent upon the availability of the renal hormone 1,25-(OH)2-D in vitamin D-replete humans when dietary Ca intake is normal. By contrast, other factors must play a dominant role in regulating net intestinal PO4 and Mg absorption.
Additional Links: PMID-484524
Publisher:
PubMed:
Citation:
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@article {pmid484524,
year = {1979},
author = {Wilz, DR and Gray, RW and Dominguez, JH and Lemann, J},
title = {Plasma 1,25-(OH)2-vitamin D concentrations and net intestinal calcium, phosphate, and magnesium absorption in humans.},
journal = {The American journal of clinical nutrition},
volume = {32},
number = {10},
pages = {2052-2060},
doi = {10.1093/ajcn/32.10.2052},
pmid = {484524},
issn = {0002-9165},
mesh = {Adult ; Calcium/*metabolism ; Dihydroxycholecalciferols/*blood ; Feces/analysis ; Female ; Humans ; Hydroxycholecalciferols/*blood ; Intestinal Absorption ; Kidney/physiology ; Kidney Calculi/metabolism ; Magnesium/*metabolism ; Male ; Middle Aged ; Nephrectomy ; Phosphates/*metabolism ; Renal Dialysis ; },
abstract = {We evaluated the relationship between plasma concentrations of the renal hormone 1,25-(OH)2-vitamin D and net intestinal absorption of Ca, PO4, and Mg in vitamin D-replete patients eating similar diets, who had undetectable, normal or elevated plasma 1,25-(OH)2-D levels, Net intestinal Ca absorption was positively correlated to plasma 1,25-(OH)2-D concentrations: percentage dietary Ca absorbed = 10 + 0.17 x plasma total 1,25-(OH)2-3, pmole/liter, r = + 0.58; P less than 0.001. By contrast, there was no significant correlation between PO4 or Mg absorption and plasma 1,25-(OH)2-D concentrations. Moreover, significant quantities of PO4 and Mg were absorbed in the absence of detectable plasma 1,25-(OH)2-D. We conclude that net intestinal Ca absorption is critically dependent upon the availability of the renal hormone 1,25-(OH)2-D in vitamin D-replete humans when dietary Ca intake is normal. By contrast, other factors must play a dominant role in regulating net intestinal PO4 and Mg absorption.},
}
MeSH Terms:
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Adult
Calcium/*metabolism
Dihydroxycholecalciferols/*blood
Feces/analysis
Female
Humans
Hydroxycholecalciferols/*blood
Intestinal Absorption
Kidney/physiology
Kidney Calculi/metabolism
Magnesium/*metabolism
Male
Middle Aged
Nephrectomy
Phosphates/*metabolism
Renal Dialysis
RevDate: 2013-11-21
CmpDate: 1979-12-27
Disorders of calcium and phosphate metabolism in childhood and adolescence.
Major problems in clinical pediatrics, 20:1-314.
Additional Links: PMID-491744
PubMed:
Citation:
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@article {pmid491744,
year = {1979},
author = {Harrison, HE and Harrison, HC},
title = {Disorders of calcium and phosphate metabolism in childhood and adolescence.},
journal = {Major problems in clinical pediatrics},
volume = {20},
number = {},
pages = {1-314},
pmid = {491744},
issn = {0076-2865},
mesh = {Adolescent ; Animals ; Bone Diseases, Metabolic ; Bone and Bones/metabolism ; Calcinosis ; Calcium/metabolism ; Calcium Metabolism Disorders/diagnosis/*therapy ; Child ; Child, Preschool ; Female ; Homeostasis ; Humans ; Hypercalcemia ; Hypocalcemia ; Infant ; Infant, Newborn ; Male ; Osteomalacia ; Osteosclerosis ; Phosphates/*metabolism ; Phosphorus Metabolism Disorders/diagnosis/*therapy ; Pregnancy ; Rickets ; Urinary Calculi ; },
}
MeSH Terms:
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Adolescent
Animals
Bone Diseases, Metabolic
Bone and Bones/metabolism
Calcinosis
Calcium/metabolism
Calcium Metabolism Disorders/diagnosis/*therapy
Child
Child, Preschool
Female
Homeostasis
Humans
Hypercalcemia
Hypocalcemia
Infant
Infant, Newborn
Male
Osteomalacia
Osteosclerosis
Phosphates/*metabolism
Phosphorus Metabolism Disorders/diagnosis/*therapy
Pregnancy
Rickets
Urinary Calculi
RevDate: 2016-11-23
CmpDate: 1980-01-19
Idiopathic hypercalciuria with bilateral macular colobomata: a new variant of oculo-renal syndrome.
Helvetica paediatrica acta, 34(3):257-269.
Two siblings from a consanguineous family, suffering from nephrocalcinosis and nephrolithiasis caused by idiopathic hypercalciuria are described. The condition is associated with bilateral macular colobomata and tapeto-retinal degeneration. It is known that the latter can occur together with different nephropathies; however, until now it has never been described in combination with idiopathic hypercalciuria. Blood calcium levels were found to be normal, calcium excretion rates were, with one exception, more than 6 mg/kg/24 h corrected for 100 ml GFR. Hypomagnesemia of 1.5 and 1.2 mg/dl and hyermagnesuria of 1.9 and 2.5 mg/kg/24 h corrected for 100 ml GFR were found in both patients. Tubular phosphate reabsorption reached 87% and 84% at serum parathormone levels of 0.34 microgram/l and 0.31 microgram/l in the two patients, respectively. Under calcium and magnesium loading the clearance rates of calcium and magnesium were raised whilst there was only a small insignificant increase in the blood levels of these cations. Acid-base titrations showed normal excretion rates of acid and base in one patient and a mild proximal tubular acidosis in the other. Quantitative investigation of the renal concentrating and diluting capacity established a decrease in the formation of the medullary concentrating gradient in both patients.
Additional Links: PMID-500385
PubMed:
Citation:
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@article {pmid500385,
year = {1979},
author = {Meier, W and Blumberg, A and Imahorn, W and De Luca, F and Wildberger, H and Oetliker, O},
title = {Idiopathic hypercalciuria with bilateral macular colobomata: a new variant of oculo-renal syndrome.},
journal = {Helvetica paediatrica acta},
volume = {34},
number = {3},
pages = {257-269},
pmid = {500385},
issn = {0018-022X},
mesh = {Adolescent ; Calcium/blood/*urine ; *Calcium Metabolism Disorders/complications/genetics/urine ; Child ; *Coloboma/complications ; Female ; Humans ; Kidney Calculi/diagnostic imaging/genetics ; Kidney Function Tests ; Magnesium/metabolism ; Male ; Nephrocalcinosis/diagnostic imaging/genetics ; *Oculocerebrorenal Syndrome/classification ; Parathyroid Hormone/blood ; Radiography ; *Renal Tubular Transport, Inborn Errors/classification ; *Retinal Degeneration/complications/genetics/pathology ; Syndrome ; },
abstract = {Two siblings from a consanguineous family, suffering from nephrocalcinosis and nephrolithiasis caused by idiopathic hypercalciuria are described. The condition is associated with bilateral macular colobomata and tapeto-retinal degeneration. It is known that the latter can occur together with different nephropathies; however, until now it has never been described in combination with idiopathic hypercalciuria. Blood calcium levels were found to be normal, calcium excretion rates were, with one exception, more than 6 mg/kg/24 h corrected for 100 ml GFR. Hypomagnesemia of 1.5 and 1.2 mg/dl and hyermagnesuria of 1.9 and 2.5 mg/kg/24 h corrected for 100 ml GFR were found in both patients. Tubular phosphate reabsorption reached 87% and 84% at serum parathormone levels of 0.34 microgram/l and 0.31 microgram/l in the two patients, respectively. Under calcium and magnesium loading the clearance rates of calcium and magnesium were raised whilst there was only a small insignificant increase in the blood levels of these cations. Acid-base titrations showed normal excretion rates of acid and base in one patient and a mild proximal tubular acidosis in the other. Quantitative investigation of the renal concentrating and diluting capacity established a decrease in the formation of the medullary concentrating gradient in both patients.},
}
MeSH Terms:
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Adolescent
Calcium/blood/*urine
*Calcium Metabolism Disorders/complications/genetics/urine
Child
*Coloboma/complications
Female
Humans
Kidney Calculi/diagnostic imaging/genetics
Kidney Function Tests
Magnesium/metabolism
Male
Nephrocalcinosis/diagnostic imaging/genetics
*Oculocerebrorenal Syndrome/classification
Parathyroid Hormone/blood
Radiography
*Renal Tubular Transport, Inborn Errors/classification
*Retinal Degeneration/complications/genetics/pathology
Syndrome
RevDate: 2019-09-11
CmpDate: 1980-03-24
Influence of an orally administered calcium-binding cation exchanger on calcium metabolism in the rat.
Urological research, 7(4):277-279.
Additional Links: PMID-524565
PubMed:
Citation:
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@article {pmid524565,
year = {1979},
author = {Hagmaier, V and Flückiger, B and Scholer, A and Remagen, W and Rutishauser, G},
title = {Influence of an orally administered calcium-binding cation exchanger on calcium metabolism in the rat.},
journal = {Urological research},
volume = {7},
number = {4},
pages = {277-279},
pmid = {524565},
issn = {0300-5623},
mesh = {Administration, Oral ; Animals ; Bone and Bones/metabolism ; Calcium/*metabolism ; Cation Exchange Resins/*administration & dosage/pharmacology ; Diet ; Female ; Ion Exchange Resins/*administration & dosage ; Rats ; Urinary Calculi/prevention & control ; },
}
MeSH Terms:
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Administration, Oral
Animals
Bone and Bones/metabolism
Calcium/*metabolism
Cation Exchange Resins/*administration & dosage/pharmacology
Diet
Female
Ion Exchange Resins/*administration & dosage
Rats
Urinary Calculi/prevention & control
RevDate: 2016-11-23
CmpDate: 1980-03-24
[Parathyroid adenoma and renal lithiasis. Report of a case].
Archivos espanoles de urologia, 32(6):569-580.
A case is presented of nephrolithiasis in a patient with no other symptoms than the urological ones and in which considerable hypercalcemia led to a study being carried out on his phospho-calcium metabolism and the diagnosis reached was primary hyperparathyroidism caused by a parathyroid adenoma. Surgical treatment was performed on the lithiasis and the adenoma as a result of which the symptoms completely disappeared and the biochemical readings returned to normal.
Additional Links: PMID-526057
PubMed:
Citation:
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@article {pmid526057,
year = {1979},
author = {Perales Juan, JL and Ferrutxe Frau, J and Llopis MÃnguez, B},
title = {[Parathyroid adenoma and renal lithiasis. Report of a case].},
journal = {Archivos espanoles de urologia},
volume = {32},
number = {6},
pages = {569-580},
pmid = {526057},
issn = {0004-0614},
mesh = {Adenoma/*complications/diagnostic imaging/surgery ; Humans ; Hypercalcemia/etiology ; Hyperparathyroidism/diagnostic imaging/*etiology ; Kidney Calculi/*complications/diagnostic imaging/surgery ; Middle Aged ; Parathyroid Neoplasms/*complications/diagnostic imaging/surgery ; Urography ; },
abstract = {A case is presented of nephrolithiasis in a patient with no other symptoms than the urological ones and in which considerable hypercalcemia led to a study being carried out on his phospho-calcium metabolism and the diagnosis reached was primary hyperparathyroidism caused by a parathyroid adenoma. Surgical treatment was performed on the lithiasis and the adenoma as a result of which the symptoms completely disappeared and the biochemical readings returned to normal.},
}
MeSH Terms:
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Adenoma/*complications/diagnostic imaging/surgery
Humans
Hypercalcemia/etiology
Hyperparathyroidism/diagnostic imaging/*etiology
Kidney Calculi/*complications/diagnostic imaging/surgery
Middle Aged
Parathyroid Neoplasms/*complications/diagnostic imaging/surgery
Urography
RevDate: 2017-11-17
CmpDate: 1980-05-30
The influence of glucose and crude fibre (wheat bran) on the rate of intestinal 47Ca absorption. The influence of glucose and wheat bran on calcium absorption.
Journal of the Royal Naval Medical Service, 65(3):143-146.
Additional Links: PMID-536945
PubMed:
Citation:
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@article {pmid536945,
year = {1979},
author = {Macleod, MA and Blacklock, NJ},
title = {The influence of glucose and crude fibre (wheat bran) on the rate of intestinal 47Ca absorption. The influence of glucose and wheat bran on calcium absorption.},
journal = {Journal of the Royal Naval Medical Service},
volume = {65},
number = {3},
pages = {143-146},
pmid = {536945},
issn = {0035-9033},
mesh = {Absorption ; Calcium/*metabolism ; Calcium Oxalate/metabolism ; Calcium Radioisotopes ; *Cellulose ; *Dietary Fiber ; Glucose/*pharmacology ; Humans ; Intestinal Absorption/*drug effects ; Kidney Calculi/metabolism ; Phytic Acid/pharmacology ; *Triticum ; },
}
MeSH Terms:
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Absorption
Calcium/*metabolism
Calcium Oxalate/metabolism
Calcium Radioisotopes
*Cellulose
*Dietary Fiber
Glucose/*pharmacology
Humans
Intestinal Absorption/*drug effects
Kidney Calculi/metabolism
Phytic Acid/pharmacology
*Triticum
RevDate: 2013-11-21
CmpDate: 1980-06-16
[Increased incidence of nephrolithiasis among lifeguards in Israel].
Harefuah, 97(3-4):57-59.
Additional Links: PMID-540823
PubMed:
Citation:
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@article {pmid540823,
year = {1979},
author = {Shabtai, M and Malamud, A and Berenheim, J and Haimovitz, C and Kedar, SS and Better, OS},
title = {[Increased incidence of nephrolithiasis among lifeguards in Israel].},
journal = {Harefuah},
volume = {97},
number = {3-4},
pages = {57-59},
pmid = {540823},
issn = {0017-7768},
mesh = {Calcium/metabolism ; Humans ; Israel ; Kidney Calculi/*epidemiology/metabolism ; Occupational Diseases/*epidemiology ; Swimming ; },
}
MeSH Terms:
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Calcium/metabolism
Humans
Israel
Kidney Calculi/*epidemiology/metabolism
Occupational Diseases/*epidemiology
Swimming
RevDate: 2013-11-21
CmpDate: 1980-06-25
Urinary zinc excretion in recurrent calcareous nephrolithiasis (RCN) patients.
Minerva nefrologica, 26(4):537-542.
Additional Links: PMID-542223
PubMed:
Citation:
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@article {pmid542223,
year = {1979},
author = {Surian, M and Graziani, G and Colussi, G and Antonacci, A and Aroldi, A and Giacchetti, M and Masi, F and Civati, G and Minetti, L},
title = {Urinary zinc excretion in recurrent calcareous nephrolithiasis (RCN) patients.},
journal = {Minerva nefrologica},
volume = {26},
number = {4},
pages = {537-542},
pmid = {542223},
issn = {0026-4873},
mesh = {Calcium/metabolism/urine ; Humans ; Kidney Calculi/*metabolism ; Zinc/metabolism/*urine ; },
}
MeSH Terms:
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Calcium/metabolism/urine
Humans
Kidney Calculi/*metabolism
Zinc/metabolism/*urine
RevDate: 2013-11-21
CmpDate: 1978-02-18
[Etiology and pathogenesis of urolithiasis].
Zeitschrift fur arztliche Fortbildung, 71(18):856-861.
Additional Links: PMID-595633
PubMed:
Citation:
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@article {pmid595633,
year = {1977},
author = {Schneider, HJ},
title = {[Etiology and pathogenesis of urolithiasis].},
journal = {Zeitschrift fur arztliche Fortbildung},
volume = {71},
number = {18},
pages = {856-861},
pmid = {595633},
issn = {0044-2178},
mesh = {Acidosis, Renal Tubular/complications ; Adult ; Calcium/metabolism ; Crystallization ; Cystinuria/complications ; Female ; Humans ; Magnesium/metabolism ; Male ; Oxalates/metabolism ; Phosphates/metabolism ; Uric Acid/metabolism ; Urinary Calculi/epidemiology/*etiology/metabolism ; Xanthines/metabolism ; },
}
MeSH Terms:
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hide MeSH Terms
Acidosis, Renal Tubular/complications
Adult
Calcium/metabolism
Crystallization
Cystinuria/complications
Female
Humans
Magnesium/metabolism
Male
Oxalates/metabolism
Phosphates/metabolism
Uric Acid/metabolism
Urinary Calculi/epidemiology/*etiology/metabolism
Xanthines/metabolism
RevDate: 2013-11-21
CmpDate: 1978-02-18
[Significance of oxalic acid in urinary calculi].
Zeitschrift fur Urologie und Nephrologie, 70(10):757-761.
Additional Links: PMID-595926
PubMed:
Citation:
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@article {pmid595926,
year = {1977},
author = {Schneider, HJ},
title = {[Significance of oxalic acid in urinary calculi].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {70},
number = {10},
pages = {757-761},
pmid = {595926},
issn = {0044-3611},
mesh = {Binding Sites ; Calcium/metabolism ; Humans ; Intestinal Absorption ; Oxalates/*metabolism ; Postoperative Complications/metabolism ; Urinary Calculi/*metabolism/surgery ; },
}
MeSH Terms:
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Binding Sites
Calcium/metabolism
Humans
Intestinal Absorption
Oxalates/*metabolism
Postoperative Complications/metabolism
Urinary Calculi/*metabolism/surgery
RevDate: 2013-11-21
CmpDate: 1978-05-17
Effect of virus dietary mineral concentrations on the occurrence of feline urolithiasis.
Journal of the American Veterinary Medical Association, 172(5):559-563.
Additional Links: PMID-632192
PubMed:
Citation:
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@article {pmid632192,
year = {1978},
author = {Lewis, LD and Chow, FH and Taton, GF and Hamar, DW},
title = {Effect of virus dietary mineral concentrations on the occurrence of feline urolithiasis.},
journal = {Journal of the American Veterinary Medical Association},
volume = {172},
number = {5},
pages = {559-563},
pmid = {632192},
issn = {0003-1488},
mesh = {Animal Feed ; Animals ; Calcium/metabolism ; Cat Diseases/etiology/*metabolism ; Cats ; Kidney/metabolism ; Magnesium/metabolism ; Male ; Minerals/*metabolism ; Phosphates/metabolism ; Urinary Bladder/metabolism ; Urinary Calculi/etiology/metabolism/*veterinary ; },
}
MeSH Terms:
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Animal Feed
Animals
Calcium/metabolism
Cat Diseases/etiology/*metabolism
Cats
Kidney/metabolism
Magnesium/metabolism
Male
Minerals/*metabolism
Phosphates/metabolism
Urinary Bladder/metabolism
Urinary Calculi/etiology/metabolism/*veterinary
RevDate: 2022-03-18
CmpDate: 1978-06-28
Thiazides and calcium urolithiasis.
Canadian Medical Association journal, 118(7):755-758.
Additional Links: PMID-638900
PubMed:
Citation:
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@article {pmid638900,
year = {1978},
author = {Yendt, ER and Cohanim, M},
title = {Thiazides and calcium urolithiasis.},
journal = {Canadian Medical Association journal},
volume = {118},
number = {7},
pages = {755-758},
pmid = {638900},
issn = {0008-4409},
mesh = {Calcium/metabolism ; Diuretics ; Humans ; Hydrochlorothiazide/therapeutic use ; Kidney Calculi/*drug therapy/metabolism/prevention & control ; Recurrence ; Sodium Chloride Symporter Inhibitors/*therapeutic use ; },
}
MeSH Terms:
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Calcium/metabolism
Diuretics
Humans
Hydrochlorothiazide/therapeutic use
Kidney Calculi/*drug therapy/metabolism/prevention & control
Recurrence
Sodium Chloride Symporter Inhibitors/*therapeutic use
RevDate: 2013-11-21
CmpDate: 1978-07-15
Kinetics of early time calcium oxalate nephrolithiasis.
Investigative urology, 15(6):465-468.
This paper examines the kinetics of calcium deposition in rat kidneys after an intraperitoneal sodium oxalate injection. From the results we conclude that only a limited portion of tubular surface is available for adsorption of calcium oxalate crystals, that adsorpption of calcium oxalate crystals onto tubular epithelium is a process of greater than first order with regard to the dose, and that the washout of retained particles from the tubules is a first-order process as related to time. Also, we conclude that in these animals, which were subjected to a large oxalate challenge, the deposition of calcium oxalate crystals is virtually all intratubular.
Additional Links: PMID-649295
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Citation:
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@article {pmid649295,
year = {1978},
author = {Jordan, WR and Finlayson, B and Luxenberg, M},
title = {Kinetics of early time calcium oxalate nephrolithiasis.},
journal = {Investigative urology},
volume = {15},
number = {6},
pages = {465-468},
pmid = {649295},
issn = {0021-0005},
mesh = {Animals ; Calcium/*metabolism ; Disease Models, Animal ; Dose-Response Relationship, Drug ; Kidney/*metabolism/pathology ; Kidney Calculi/*metabolism/pathology ; Male ; Oxalates/*metabolism/pharmacology ; Rats ; Sodium/pharmacology ; },
abstract = {This paper examines the kinetics of calcium deposition in rat kidneys after an intraperitoneal sodium oxalate injection. From the results we conclude that only a limited portion of tubular surface is available for adsorption of calcium oxalate crystals, that adsorpption of calcium oxalate crystals onto tubular epithelium is a process of greater than first order with regard to the dose, and that the washout of retained particles from the tubules is a first-order process as related to time. Also, we conclude that in these animals, which were subjected to a large oxalate challenge, the deposition of calcium oxalate crystals is virtually all intratubular.},
}
MeSH Terms:
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Animals
Calcium/*metabolism
Disease Models, Animal
Dose-Response Relationship, Drug
Kidney/*metabolism/pathology
Kidney Calculi/*metabolism/pathology
Male
Oxalates/*metabolism/pharmacology
Rats
Sodium/pharmacology
RevDate: 2013-11-21
CmpDate: 1978-07-24
[Kidney calculi--current viewpoints on diagnosis and therapy].
Lakartidningen, 75(19):1909-1910.
Additional Links: PMID-651448
PubMed:
Citation:
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@article {pmid651448,
year = {1978},
author = {Fritjofsson, A},
title = {[Kidney calculi--current viewpoints on diagnosis and therapy].},
journal = {Lakartidningen},
volume = {75},
number = {19},
pages = {1909-1910},
pmid = {651448},
issn = {0023-7205},
mesh = {Calcium/metabolism ; Humans ; *Kidney Calculi/diagnosis/metabolism/surgery ; },
}
MeSH Terms:
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Calcium/metabolism
Humans
*Kidney Calculi/diagnosis/metabolism/surgery
RevDate: 2013-11-21
CmpDate: 1978-07-24
[Calcium metabolism disorders in kidney calculi patients].
Lakartidningen, 75(19):1911-1912.
Additional Links: PMID-651449
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@article {pmid651449,
year = {1978},
author = {Berlin, T and Collste, L and von Garrelts, B},
title = {[Calcium metabolism disorders in kidney calculi patients].},
journal = {Lakartidningen},
volume = {75},
number = {19},
pages = {1911-1912},
pmid = {651449},
issn = {0023-7205},
mesh = {Bendroflumethiazide/therapeutic use ; Calcium Metabolism Disorders/*complications ; Female ; Humans ; Kidney Calculi/*metabolism/prevention & control ; Male ; Recurrence ; },
}
MeSH Terms:
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Bendroflumethiazide/therapeutic use
Calcium Metabolism Disorders/*complications
Female
Humans
Kidney Calculi/*metabolism/prevention & control
Male
Recurrence
RevDate: 2019-05-09
CmpDate: 1978-07-15
Microliths in sputum specimens and their relationship to pulmonary alveolar microlithiasis.
American journal of clinical pathology, 69(5):482-485.
The sputa of 26 of 100 patients with chronic obstructive pulmonary disease contained microliths. The numbers of microliths per smear of sputum per patient ranged from one to 20. In seven of the sputum specimens containing microliths, deposition of calcium was present on the loops of Curshmann's spirals to different extents, from a single layer to the laminated appearance seen in a free microlith. Partial and complete separation of the calcified laminated masses from the spirals were also demonstrated. Based on these observations, the morphogenesis of microliths and the pathogenesis of pulmonary alveolar microlithiasis are discussed.
Additional Links: PMID-655126
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@article {pmid655126,
year = {1978},
author = {Tao, LC},
title = {Microliths in sputum specimens and their relationship to pulmonary alveolar microlithiasis.},
journal = {American journal of clinical pathology},
volume = {69},
number = {5},
pages = {482-485},
doi = {10.1093/ajcp/69.5.482},
pmid = {655126},
issn = {0002-9173},
mesh = {Calcium/metabolism ; Calculi/diagnosis/*etiology ; Female ; Humans ; Lung Diseases, Obstructive/*complications ; Male ; Middle Aged ; Mucus ; *Pulmonary Alveoli ; *Sputum ; },
abstract = {The sputa of 26 of 100 patients with chronic obstructive pulmonary disease contained microliths. The numbers of microliths per smear of sputum per patient ranged from one to 20. In seven of the sputum specimens containing microliths, deposition of calcium was present on the loops of Curshmann's spirals to different extents, from a single layer to the laminated appearance seen in a free microlith. Partial and complete separation of the calcified laminated masses from the spirals were also demonstrated. Based on these observations, the morphogenesis of microliths and the pathogenesis of pulmonary alveolar microlithiasis are discussed.},
}
MeSH Terms:
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Calcium/metabolism
Calculi/diagnosis/*etiology
Female
Humans
Lung Diseases, Obstructive/*complications
Male
Middle Aged
Mucus
*Pulmonary Alveoli
*Sputum
RevDate: 2013-11-21
CmpDate: 1978-09-25
[Investigation of the causes of urolithiasts. I. Calcium metabolism, with special reference to serum ionized calcium (author's transl)].
Nihon Hinyokika Gakkai zasshi. The japanese journal of urology, 69(5):578-584.
Additional Links: PMID-671914
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@article {pmid671914,
year = {1978},
author = {Iguchi, M and Nagai, N and Matsuura, T and Kaneko, S and Kohri, K and Minami, K and Kadowaki, T and Akiyama, T and Yachiku, S and Kurita, T and Nakamoto, J and Ohba, Y},
title = {[Investigation of the causes of urolithiasts. I. Calcium metabolism, with special reference to serum ionized calcium (author's transl)].},
journal = {Nihon Hinyokika Gakkai zasshi. The japanese journal of urology},
volume = {69},
number = {5},
pages = {578-584},
pmid = {671914},
issn = {0021-5287},
mesh = {Adult ; Calcium/*blood ; Female ; Humans ; Male ; Middle Aged ; Urinary Calculi/*etiology ; },
}
MeSH Terms:
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Adult
Calcium/*blood
Female
Humans
Male
Middle Aged
Urinary Calculi/*etiology
RevDate: 2013-11-21
CmpDate: 1978-11-18
Renal disease and disorders of calcium metabolism.
Comprehensive therapy, 4(8):44-50.
Additional Links: PMID-688743
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@article {pmid688743,
year = {1978},
author = {Kanis, JA},
title = {Renal disease and disorders of calcium metabolism.},
journal = {Comprehensive therapy},
volume = {4},
number = {8},
pages = {44-50},
pmid = {688743},
issn = {0098-8243},
mesh = {Bone Diseases/*etiology/therapy ; Calcium/metabolism ; Calcium Metabolism Disorders/*complications ; Humans ; Kidney/metabolism ; Kidney Calculi/*etiology/therapy ; Kidney Diseases/*complications/metabolism/therapy ; Parathyroid Hormone/metabolism ; Phosphates/metabolism ; Vitamin D/metabolism ; },
}
MeSH Terms:
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Bone Diseases/*etiology/therapy
Calcium/metabolism
Calcium Metabolism Disorders/*complications
Humans
Kidney/metabolism
Kidney Calculi/*etiology/therapy
Kidney Diseases/*complications/metabolism/therapy
Parathyroid Hormone/metabolism
Phosphates/metabolism
Vitamin D/metabolism
RevDate: 2013-11-21
CmpDate: 1978-11-29
[Effect of hydrochlorothiazide on calcium-magnesium parameters in patients with recurrent nephrolithiasis].
Polski tygodnik lekarski (Warsaw, Poland : 1960), 33(30):1173-1175.
Additional Links: PMID-693386
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@article {pmid693386,
year = {1978},
author = {Musialik, D and Głuszek, J and Raszeja-Wanic, B},
title = {[Effect of hydrochlorothiazide on calcium-magnesium parameters in patients with recurrent nephrolithiasis].},
journal = {Polski tygodnik lekarski (Warsaw, Poland : 1960)},
volume = {33},
number = {30},
pages = {1173-1175},
pmid = {693386},
issn = {0032-3756},
mesh = {Adult ; Calcium/*metabolism ; Female ; Humans ; Hydrochlorothiazide/*pharmacology ; Kidney Calculi/*metabolism ; Magnesium/*metabolism ; Male ; Middle Aged ; Recurrence ; },
}
MeSH Terms:
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Adult
Calcium/*metabolism
Female
Humans
Hydrochlorothiazide/*pharmacology
Kidney Calculi/*metabolism
Magnesium/*metabolism
Male
Middle Aged
Recurrence
RevDate: 2013-11-21
CmpDate: 1978-11-18
[Effect of a calcium-deficient, phosphate- or ammonia-rich diet on the development and mineral metabolism of growing miniature pigs].
Zeitschrift fur Urologie und Nephrologie, 71(7):463-472.
The metaphylaxis of renal calculi in form of partial withdrawal of Ca and the nutrition rich in phosphate or NH4C1 in children was disputed and not ascertained by animal experiments. For this purpose growing female dwarf pigs got a semisynthetic ration with 1 g Ca and 23 g P, respectively, or 15 g NH4C1 per kg dry feeding mass during 168 days. The dwarf pigs fed poor in Ca had a reduced increase of body-weight of 30%, the pigs fed rich in P and NH4C1 a reduced increase of body-weight of 11 and 16%, respectively, compared with the control dwarf pigs. The feeding regime chosen has no significant influence on the Ca-, P-, Cu-, and Mn-state of the dwarf pigs (alpha greater than 0.05). Only the animals with deficiency of a Ca statistically ascertained stored more Zn in the liver than the control animals. The applied NH4C1 as well as the medicament Ammonchlor SSW contained traces of Cd. Concerning the lacking significant changes of metabolism on account of the retardation of growth conditioned by therapy it cannot be concluded that there is possible a metaphylaxis of the lithiasis in children by partial withdrawal of Ca or additions of P and NH4C1, respectively.
Additional Links: PMID-696021
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@article {pmid696021,
year = {1978},
author = {Anke, M and Schneider, HJ and Partschefeld, M and Grün, M},
title = {[Effect of a calcium-deficient, phosphate- or ammonia-rich diet on the development and mineral metabolism of growing miniature pigs].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {71},
number = {7},
pages = {463-472},
pmid = {696021},
issn = {0044-3611},
mesh = {Ammonia/*administration & dosage/metabolism ; Animal Nutritional Physiological Phenomena ; Animals ; Body Weight ; Brain/metabolism ; Calcium/metabolism ; Calcium, Dietary/*administration & dosage ; Copper/metabolism ; Liver/metabolism ; Manganese/metabolism ; *Nutritional Physiological Phenomena ; Phosphates/*administration & dosage/metabolism ; Species Specificity ; Swine/*growth & development ; Zinc/metabolism ; },
abstract = {The metaphylaxis of renal calculi in form of partial withdrawal of Ca and the nutrition rich in phosphate or NH4C1 in children was disputed and not ascertained by animal experiments. For this purpose growing female dwarf pigs got a semisynthetic ration with 1 g Ca and 23 g P, respectively, or 15 g NH4C1 per kg dry feeding mass during 168 days. The dwarf pigs fed poor in Ca had a reduced increase of body-weight of 30%, the pigs fed rich in P and NH4C1 a reduced increase of body-weight of 11 and 16%, respectively, compared with the control dwarf pigs. The feeding regime chosen has no significant influence on the Ca-, P-, Cu-, and Mn-state of the dwarf pigs (alpha greater than 0.05). Only the animals with deficiency of a Ca statistically ascertained stored more Zn in the liver than the control animals. The applied NH4C1 as well as the medicament Ammonchlor SSW contained traces of Cd. Concerning the lacking significant changes of metabolism on account of the retardation of growth conditioned by therapy it cannot be concluded that there is possible a metaphylaxis of the lithiasis in children by partial withdrawal of Ca or additions of P and NH4C1, respectively.},
}
MeSH Terms:
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Ammonia/*administration & dosage/metabolism
Animal Nutritional Physiological Phenomena
Animals
Body Weight
Brain/metabolism
Calcium/metabolism
Calcium, Dietary/*administration & dosage
Copper/metabolism
Liver/metabolism
Manganese/metabolism
*Nutritional Physiological Phenomena
Phosphates/*administration & dosage/metabolism
Species Specificity
Swine/*growth & development
Zinc/metabolism
RevDate: 2013-11-21
CmpDate: 1978-12-20
Properties and biosynthesis of a vitamin K-dependent calcium binding protein in bone.
Federation proceedings, 37(12):2615-2620.
Bone, whether of endochondral or intramembranous origin, contains the vitamin K-dependent calcium binding amino acid residue gamma-carboxyglutamic acid (Gla) in a small, anionic protein we have named osteocalcin. This protein, which constitutes 0.5 to 1.0% of the total bone proteins and about 20% of the noncollagenous protein, is extractable by EDTA, and contains at least 90% of bone Gla. Analysis of purified osteocalcin from chicken bone and independent sequence studies of an analogous bovine bone protein show no apparent homology to the Gla-containing region of prothrombin. Chicken osteocalcin specifically binds 2 moles of calcium ions per 6,500 g protein. Employing coumarin drugs and vitamin K-deficient diets, vitamin K-dependent osteocalcin biosynthesis has been demonstrated in the chick before and after hatching. Organ cultures of embryonic chick bone show de novo synthesis of osteocalcin, and microsomal preparations of embryonic bone also exhibit vitamin K-dependent carboxylase activity. In addition to the presence of osteocalcin in bone, a Gla-protein of unknown function is present in normal nonmineralized kidney cortex. Furthermore, in various pathological calcifications such as hard atheromatous plaque, renal calculi, and subcutaneous ectopic calcifications other Gla-proteins are found, thus implicating such proteins and vitamin K in many facets of calcium metabolism.
Additional Links: PMID-700171
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@article {pmid700171,
year = {1978},
author = {Lian, JB and Hauschka, PV and Gallop, PM},
title = {Properties and biosynthesis of a vitamin K-dependent calcium binding protein in bone.},
journal = {Federation proceedings},
volume = {37},
number = {12},
pages = {2615-2620},
pmid = {700171},
issn = {0014-9446},
mesh = {1-Carboxyglutamic Acid/metabolism ; Animals ; Bone Development ; Bone and Bones/*metabolism ; Calcification, Physiologic ; Calcinosis/metabolism ; Calcium/*metabolism ; Carboxy-Lyases/metabolism ; Carrier Proteins/*metabolism ; Chickens ; Culture Techniques ; Microsomes/enzymology ; Vitamin K/*metabolism ; },
abstract = {Bone, whether of endochondral or intramembranous origin, contains the vitamin K-dependent calcium binding amino acid residue gamma-carboxyglutamic acid (Gla) in a small, anionic protein we have named osteocalcin. This protein, which constitutes 0.5 to 1.0% of the total bone proteins and about 20% of the noncollagenous protein, is extractable by EDTA, and contains at least 90% of bone Gla. Analysis of purified osteocalcin from chicken bone and independent sequence studies of an analogous bovine bone protein show no apparent homology to the Gla-containing region of prothrombin. Chicken osteocalcin specifically binds 2 moles of calcium ions per 6,500 g protein. Employing coumarin drugs and vitamin K-deficient diets, vitamin K-dependent osteocalcin biosynthesis has been demonstrated in the chick before and after hatching. Organ cultures of embryonic chick bone show de novo synthesis of osteocalcin, and microsomal preparations of embryonic bone also exhibit vitamin K-dependent carboxylase activity. In addition to the presence of osteocalcin in bone, a Gla-protein of unknown function is present in normal nonmineralized kidney cortex. Furthermore, in various pathological calcifications such as hard atheromatous plaque, renal calculi, and subcutaneous ectopic calcifications other Gla-proteins are found, thus implicating such proteins and vitamin K in many facets of calcium metabolism.},
}
MeSH Terms:
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1-Carboxyglutamic Acid/metabolism
Animals
Bone Development
Bone and Bones/*metabolism
Calcification, Physiologic
Calcinosis/metabolism
Calcium/*metabolism
Carboxy-Lyases/metabolism
Carrier Proteins/*metabolism
Chickens
Culture Techniques
Microsomes/enzymology
Vitamin K/*metabolism
RevDate: 2013-11-21
CmpDate: 1979-02-23
[Hypercalciuria and kidney calculi].
Nederlands tijdschrift voor geneeskunde, 122(51):2020-2025.
Additional Links: PMID-723991
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@article {pmid723991,
year = {1978},
author = {Netelenbos, JC and Haagsma-Schouten, WA and Lopes Cardozo, E},
title = {[Hypercalciuria and kidney calculi].},
journal = {Nederlands tijdschrift voor geneeskunde},
volume = {122},
number = {51},
pages = {2020-2025},
pmid = {723991},
issn = {0028-2162},
mesh = {Calcitonin/physiology ; Calcium/metabolism/*urine ; Dihydroxycholecalciferols/metabolism ; Gonadal Steroid Hormones/physiology ; Growth Hormone/metabolism ; Humans ; Hydrocortisone/metabolism ; Intestine, Small/metabolism ; Kidney/metabolism ; Kidney Calculi/drug therapy/*metabolism ; Parathyroid Hormone/physiology ; Recurrence ; Thyroid Hormones/physiology ; },
}
MeSH Terms:
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Calcitonin/physiology
Calcium/metabolism/*urine
Dihydroxycholecalciferols/metabolism
Gonadal Steroid Hormones/physiology
Growth Hormone/metabolism
Humans
Hydrocortisone/metabolism
Intestine, Small/metabolism
Kidney/metabolism
Kidney Calculi/drug therapy/*metabolism
Parathyroid Hormone/physiology
Recurrence
Thyroid Hormones/physiology
RevDate: 2013-11-21
CmpDate: 1979-04-25
[Sponge kidney (review of 34 cases of a lithiasic population)].
Revista clinica espanola, 151(5):389-393.
Additional Links: PMID-741025
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@article {pmid741025,
year = {1978},
author = {Castrillo GarcÃa, J and RodrÃguez-Miñón Cifuentes, J and Salinas Casado, J},
title = {[Sponge kidney (review of 34 cases of a lithiasic population)].},
journal = {Revista clinica espanola},
volume = {151},
number = {5},
pages = {389-393},
pmid = {741025},
issn = {0014-2565},
mesh = {Adolescent ; Adult ; Aged ; Calcium/metabolism ; Diagnosis, Differential ; Female ; Humans ; Kidney Calculi/complications/*metabolism ; Male ; Medullary Sponge Kidney/complications/*diagnosis/metabolism ; Middle Aged ; Phosphorus/metabolism ; },
}
MeSH Terms:
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Adolescent
Adult
Aged
Calcium/metabolism
Diagnosis, Differential
Female
Humans
Kidney Calculi/complications/*metabolism
Male
Medullary Sponge Kidney/complications/*diagnosis/metabolism
Middle Aged
Phosphorus/metabolism
RevDate: 2019-10-21
CmpDate: 1979-07-16
Calcium urolithiasis: is it analogous to bone formation?.
Calcified tissue research, 26(3):195-197.
Additional Links: PMID-750062
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@article {pmid750062,
year = {1978},
author = {Pak, CY},
title = {Calcium urolithiasis: is it analogous to bone formation?.},
journal = {Calcified tissue research},
volume = {26},
number = {3},
pages = {195-197},
pmid = {750062},
issn = {0008-0594},
mesh = {Bone and Bones/*physiology ; Calcium/*metabolism ; Calcium Oxalate/metabolism ; Crystallization ; Humans ; Kidney/physiopathology ; Urinary Calculi/*physiopathology ; },
}
MeSH Terms:
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Bone and Bones/*physiology
Calcium/*metabolism
Calcium Oxalate/metabolism
Crystallization
Humans
Kidney/physiopathology
Urinary Calculi/*physiopathology
RevDate: 2019-08-23
CmpDate: 1979-07-16
Determination of intestinal radiocalcium absorption by double tracer method with 85Sr i.v.
European journal of nuclear medicine, 3(2):115-120.
Additional Links: PMID-750200
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@article {pmid750200,
year = {1978},
author = {Jovanović, V},
title = {Determination of intestinal radiocalcium absorption by double tracer method with 85Sr i.v.},
journal = {European journal of nuclear medicine},
volume = {3},
number = {2},
pages = {115-120},
pmid = {750200},
issn = {0340-6997},
mesh = {Adolescent ; Adult ; Biological Transport ; Calcium/metabolism ; Calcium Radioisotopes/*metabolism ; Female ; Humans ; Hyperparathyroidism/metabolism ; Hyperparathyroidism, Secondary/metabolism ; *Intestinal Absorption ; Kidney Calculi/metabolism ; Male ; Middle Aged ; Models, Biological ; *Strontium Radioisotopes ; Time Factors ; },
}
MeSH Terms:
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Adolescent
Adult
Biological Transport
Calcium/metabolism
Calcium Radioisotopes/*metabolism
Female
Humans
Hyperparathyroidism/metabolism
Hyperparathyroidism, Secondary/metabolism
*Intestinal Absorption
Kidney Calculi/metabolism
Male
Middle Aged
Models, Biological
*Strontium Radioisotopes
Time Factors
RevDate: 2013-11-21
CmpDate: 1979-07-25
Experimental urolithiasis: Part III-metabolic alterations of calcium, phosphorus & oxalate using 45Ca, 32P & oxalate (U)-14C.
Indian journal of experimental biology, 16(11):1161-1163.
Additional Links: PMID-750399
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@article {pmid750399,
year = {1978},
author = {Thind, SK and Nath, R},
title = {Experimental urolithiasis: Part III-metabolic alterations of calcium, phosphorus & oxalate using 45Ca, 32P & oxalate (U)-14C.},
journal = {Indian journal of experimental biology},
volume = {16},
number = {11},
pages = {1161-1163},
pmid = {750399},
issn = {0019-5189},
mesh = {Animals ; Calcium/*metabolism ; Calcium Radioisotopes ; Carbon Radioisotopes ; Male ; Oxalates/*metabolism ; Phosphorus/*metabolism ; Phosphorus Radioisotopes ; Rats ; Urinary Calculi/*metabolism ; },
}
MeSH Terms:
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Animals
Calcium/*metabolism
Calcium Radioisotopes
Carbon Radioisotopes
Male
Oxalates/*metabolism
Phosphorus/*metabolism
Phosphorus Radioisotopes
Rats
Urinary Calculi/*metabolism
RevDate: 2013-11-21
CmpDate: 1976-08-23
[Letter; Treatment of urinary calcic lithiasis with allopurinol].
La Nouvelle presse medicale, 5(21):1363.
Additional Links: PMID-778792
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Citation:
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@article {pmid778792,
year = {1976},
author = {Berthoux, FC},
title = {[Letter; Treatment of urinary calcic lithiasis with allopurinol].},
journal = {La Nouvelle presse medicale},
volume = {5},
number = {21},
pages = {1363},
pmid = {778792},
issn = {0301-1518},
mesh = {Allopurinol/*therapeutic use ; Calcium/metabolism/urine ; Clinical Trials as Topic ; Female ; Humans ; Kidney Calculi/*drug therapy/metabolism ; Male ; Uric Acid/metabolism ; },
}
MeSH Terms:
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Allopurinol/*therapeutic use
Calcium/metabolism/urine
Clinical Trials as Topic
Female
Humans
Kidney Calculi/*drug therapy/metabolism
Male
Uric Acid/metabolism
RevDate: 2016-11-23
CmpDate: 1988-03-21
[Echographic and urographic findings in a case of liquid renal calculosis].
La Radiologia medica, 62(3):196-201.
Additional Links: PMID-802907
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@article {pmid802907,
year = {1976},
author = {Biggi, E and Rosso, F and Oliveri, M and Luzi, G},
title = {[Echographic and urographic findings in a case of liquid renal calculosis].},
journal = {La Radiologia medica},
volume = {62},
number = {3},
pages = {196-201},
pmid = {802907},
issn = {0033-8362},
mesh = {Adult ; Calcium/metabolism ; Female ; Humans ; Kidney/metabolism ; Kidney Calculi/*diagnosis/diagnostic imaging/etiology/pathology ; Kidney Diseases, Cystic/complications/*diagnosis/diagnostic imaging ; Radiography ; *Ultrasonography ; },
}
MeSH Terms:
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Adult
Calcium/metabolism
Female
Humans
Kidney/metabolism
Kidney Calculi/*diagnosis/diagnostic imaging/etiology/pathology
Kidney Diseases, Cystic/complications/*diagnosis/diagnostic imaging
Radiography
*Ultrasonography
RevDate: 2015-11-19
CmpDate: 1976-09-02
[Nephrolithiasis in hypercalciuria and hyperparathyroidism].
MMW, Munchener medizinische Wochenschrift, 118(25):799-810.
In the group of 1635 patients with nephorolithiasis the authors found 16% with idiopathic hypercalciuria. They administered thiazide diuretics for 8 days as a test for the detection of masked hyperparathyroidism in idiopathic hypercalciuria. In 6 cases which were diagnosed in this manner, the existence of a parathyroidadenoma could be demonstrated by surgical operation. The hypercalciuria persisted after exstirpation of the tumor, in some cases the nephrolithiasis also remained active. the various pathogenetic mechanisms connected with these common diseases are discussed in relation to these results.
Additional Links: PMID-819807
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@article {pmid819807,
year = {1976},
author = {Rapado, A and de la Calle, H and Castrillo, JM and Mancha, A and Traba, ML and Delatte, LC},
title = {[Nephrolithiasis in hypercalciuria and hyperparathyroidism].},
journal = {MMW, Munchener medizinische Wochenschrift},
volume = {118},
number = {25},
pages = {799-810},
pmid = {819807},
issn = {0341-3098},
mesh = {Adenoma/complications/etiology ; Adult ; Aged ; Benzothiadiazines ; Calcium/metabolism/*urine ; Diuretics ; Humans ; Hypercalcemia ; Hyperparathyroidism/*complications/diagnosis ; Middle Aged ; Parathyroid Neoplasms/complications/etiology/surgery ; Sodium Chloride Symporter Inhibitors/adverse effects/pharmacology/therapeutic use ; Urinary Calculi/*complications/drug therapy/etiology ; },
abstract = {In the group of 1635 patients with nephorolithiasis the authors found 16% with idiopathic hypercalciuria. They administered thiazide diuretics for 8 days as a test for the detection of masked hyperparathyroidism in idiopathic hypercalciuria. In 6 cases which were diagnosed in this manner, the existence of a parathyroidadenoma could be demonstrated by surgical operation. The hypercalciuria persisted after exstirpation of the tumor, in some cases the nephrolithiasis also remained active. the various pathogenetic mechanisms connected with these common diseases are discussed in relation to these results.},
}
MeSH Terms:
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hide MeSH Terms
Adenoma/complications/etiology
Adult
Aged
Benzothiadiazines
Calcium/metabolism/*urine
Diuretics
Humans
Hypercalcemia
Hyperparathyroidism/*complications/diagnosis
Middle Aged
Parathyroid Neoplasms/complications/etiology/surgery
Sodium Chloride Symporter Inhibitors/adverse effects/pharmacology/therapeutic use
Urinary Calculi/*complications/drug therapy/etiology
RevDate: 2013-11-21
CmpDate: 1977-02-16
[Parathyroid adenoma from the surgical view point. Report of 65 cases].
MMW, Munchener medizinische Wochenschrift, 118(48):1549-1554.
Experience in the surgical management of a series of 65 cases with hyperparathyroidism is described (primary HPT: 55, secondary HPT: 3, tertiary HPT: 2, malignant HPT: 5) The consecutive forms of primary HPT, i.e. quarternary or quinternary HPT, and of secondary HPT, i.e. tertiary HPT, are discussed in more detail. Dystopic location was observed in 12.3%. In 4.6% no adenoma was found during the operation. A modern method for the localisation is the measurement of parathyroid hormone levels by radioimmunoassay. In nearly all publications we observe an increase in the renal forms. Intensive search for primary HPT is essential in all cases with recurrent renal calculi.
Additional Links: PMID-826794
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@article {pmid826794,
year = {1976},
author = {Keminger, K},
title = {[Parathyroid adenoma from the surgical view point. Report of 65 cases].},
journal = {MMW, Munchener medizinische Wochenschrift},
volume = {118},
number = {48},
pages = {1549-1554},
pmid = {826794},
issn = {0341-3098},
mesh = {Adenoma/surgery ; Adolescent ; Adult ; Aged ; Calcinosis/etiology ; Calcium/metabolism ; Carcinoma/surgery ; Female ; Follow-Up Studies ; Humans ; Hyperparathyroidism/complications/*surgery ; Hyperparathyroidism, Secondary/surgery ; Male ; Middle Aged ; Parathyroid Neoplasms/*surgery ; Phosphates/blood ; Potassium/blood ; },
abstract = {Experience in the surgical management of a series of 65 cases with hyperparathyroidism is described (primary HPT: 55, secondary HPT: 3, tertiary HPT: 2, malignant HPT: 5) The consecutive forms of primary HPT, i.e. quarternary or quinternary HPT, and of secondary HPT, i.e. tertiary HPT, are discussed in more detail. Dystopic location was observed in 12.3%. In 4.6% no adenoma was found during the operation. A modern method for the localisation is the measurement of parathyroid hormone levels by radioimmunoassay. In nearly all publications we observe an increase in the renal forms. Intensive search for primary HPT is essential in all cases with recurrent renal calculi.},
}
MeSH Terms:
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hide MeSH Terms
Adenoma/surgery
Adolescent
Adult
Aged
Calcinosis/etiology
Calcium/metabolism
Carcinoma/surgery
Female
Follow-Up Studies
Humans
Hyperparathyroidism/complications/*surgery
Hyperparathyroidism, Secondary/surgery
Male
Middle Aged
Parathyroid Neoplasms/*surgery
Phosphates/blood
Potassium/blood
RevDate: 2018-11-13
CmpDate: 1977-03-31
Purpura associated with hypergammaglobulinemia, renal tubular acidosis and osteomalacia.
Canadian Medical Association journal, 116(1):53-58.
Two patients with hyperglobulinemia associated with purpura were studied. One had features of Sjögren's syndrome, while the other appeared to have a primary condition -- "chronic benign purpura". Both patients also had renal tubular acidosis, osteomalacia and renal calculi, with disturbed calcium metabolism and acid-base balance. Autoantibodies were detected in the serum of both patients, and mononuclear cell infiltrates were noted in skin and kidney biopsies from both.
Additional Links: PMID-832220
PubMed:
Citation:
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@article {pmid832220,
year = {1977},
author = {Marquez-Julio, A and Rapoport, A and Wilansky, DL and Rabinovich, S and Chamberlain, D},
title = {Purpura associated with hypergammaglobulinemia, renal tubular acidosis and osteomalacia.},
journal = {Canadian Medical Association journal},
volume = {116},
number = {1},
pages = {53-58},
pmid = {832220},
issn = {0008-4409},
mesh = {Acid-Base Equilibrium ; Acidosis, Renal Tubular/*complications/diagnosis ; Blood Proteins/analysis ; Electrolytes/blood ; Female ; Humans ; Ilium/pathology ; Immunoglobulins/analysis ; Kidney/pathology ; Kidney Function Tests ; Middle Aged ; Osteomalacia/*complications/diagnosis ; Purpura, Hyperglobulinemic/*complications/diagnosis ; Sjogren's Syndrome/complications/diagnosis ; Skin/pathology ; },
abstract = {Two patients with hyperglobulinemia associated with purpura were studied. One had features of Sjögren's syndrome, while the other appeared to have a primary condition -- "chronic benign purpura". Both patients also had renal tubular acidosis, osteomalacia and renal calculi, with disturbed calcium metabolism and acid-base balance. Autoantibodies were detected in the serum of both patients, and mononuclear cell infiltrates were noted in skin and kidney biopsies from both.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Acid-Base Equilibrium
Acidosis, Renal Tubular/*complications/diagnosis
Blood Proteins/analysis
Electrolytes/blood
Female
Humans
Ilium/pathology
Immunoglobulins/analysis
Kidney/pathology
Kidney Function Tests
Middle Aged
Osteomalacia/*complications/diagnosis
Purpura, Hyperglobulinemic/*complications/diagnosis
Sjogren's Syndrome/complications/diagnosis
Skin/pathology
RevDate: 2013-11-21
CmpDate: 1977-03-15
[The role of uric acid in calcium-oxalate nephrolithiasis].
Fortschritte der Medizin, 95(2):67.
Additional Links: PMID-832834
PubMed:
Citation:
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@article {pmid832834,
year = {1977},
author = {Hartung, R},
title = {[The role of uric acid in calcium-oxalate nephrolithiasis].},
journal = {Fortschritte der Medizin},
volume = {95},
number = {2},
pages = {67},
pmid = {832834},
issn = {0015-8178},
mesh = {Calcium/*metabolism/urine ; Crystallization ; Humans ; Kidney Calculi/*blood/metabolism/urine ; Oxalates/*metabolism/urine ; Uric Acid/*blood/urine ; },
}
MeSH Terms:
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Calcium/*metabolism/urine
Crystallization
Humans
Kidney Calculi/*blood/metabolism/urine
Oxalates/*metabolism/urine
Uric Acid/*blood/urine
RevDate: 2013-11-21
CmpDate: 1977-04-30
[Metabolic causes of nephrolithiasis].
Polski przeglad chirurgiczny, 49(2A):219-226.
Additional Links: PMID-840788
PubMed:
Citation:
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@article {pmid840788,
year = {1977},
author = {Kokot, F},
title = {[Metabolic causes of nephrolithiasis].},
journal = {Polski przeglad chirurgiczny},
volume = {49},
number = {2A},
pages = {219-226},
pmid = {840788},
issn = {0032-373X},
mesh = {Calcium/metabolism ; Cystine/metabolism ; Humans ; Kidney Calculi/*etiology/metabolism ; Models, Chemical ; Oxalates/metabolism ; Purines/metabolism ; },
}
MeSH Terms:
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Calcium/metabolism
Cystine/metabolism
Humans
Kidney Calculi/*etiology/metabolism
Models, Chemical
Oxalates/metabolism
Purines/metabolism
RevDate: 2013-11-21
CmpDate: 1977-05-20
[Idiopathic hypercalciuria and urolithiasis].
Duodecim; laaketieteellinen aikakauskirja, 93(3):195-197.
Additional Links: PMID-844442
PubMed:
Citation:
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@article {pmid844442,
year = {1977},
author = {Elomaa, I},
title = {[Idiopathic hypercalciuria and urolithiasis].},
journal = {Duodecim; laaketieteellinen aikakauskirja},
volume = {93},
number = {3},
pages = {195-197},
pmid = {844442},
issn = {0012-7183},
mesh = {Calcium/*urine ; *Calcium Metabolism Disorders ; Humans ; Urinary Calculi/*etiology ; },
}
MeSH Terms:
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Calcium/*urine
*Calcium Metabolism Disorders
Humans
Urinary Calculi/*etiology
RevDate: 2016-11-23
CmpDate: 1977-06-11
Calcium and the kidney: renal osteodystrophy and renal calculi.
Connecticut medicine, 41(4):205-209.
Additional Links: PMID-852281
PubMed:
Citation:
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@article {pmid852281,
year = {1977},
author = {Mundy, GR and Canalis, EM and Raisz, LG},
title = {Calcium and the kidney: renal osteodystrophy and renal calculi.},
journal = {Connecticut medicine},
volume = {41},
number = {4},
pages = {205-209},
pmid = {852281},
issn = {0010-6178},
mesh = {Calcium/metabolism ; *Chronic Kidney Disease-Mineral and Bone Disorder/therapy ; Humans ; *Kidney Calculi/etiology/therapy ; Kidney Failure, Chronic/complications/therapy ; Osteitis Fibrosa Cystica/etiology ; Osteomalacia/etiology ; Osteoporosis ; Osteosclerosis/complications ; Phosphates/blood ; Recurrence ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/metabolism
*Chronic Kidney Disease-Mineral and Bone Disorder/therapy
Humans
*Kidney Calculi/etiology/therapy
Kidney Failure, Chronic/complications/therapy
Osteitis Fibrosa Cystica/etiology
Osteomalacia/etiology
Osteoporosis
Osteosclerosis/complications
Phosphates/blood
Recurrence
RevDate: 2013-11-21
CmpDate: 1977-08-12
[Topographic phase analysis of urinary concrements using crystal-optic methods].
Zeitschrift fur Urologie und Nephrologie, 70(2):99-105.
In the present paper was reported on the results of the topographic crystal-optic analysis of urinary calculi of 560 concrements. 59% of all urinary calculi had a different phase content in the nucleus and in the calyx. All frequent minerals of the urinary calculi could be proved in the nucleus of the calculus. It could be shown that nucleus and calyx may have a monomineral as well as a polymineral structure. It is referred to the importance of the topographic analysis of the urinary calculi for the metaphylaxis of the urolithiasis.
Additional Links: PMID-878682
PubMed:
Citation:
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@article {pmid878682,
year = {1977},
author = {Bick, C and Schubert, G and Brien, G and Gremske, D},
title = {[Topographic phase analysis of urinary concrements using crystal-optic methods].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {70},
number = {2},
pages = {99-105},
pmid = {878682},
issn = {0044-3611},
mesh = {Calcium/metabolism ; Humans ; Methods ; *Optics and Photonics ; Oxalates/metabolism ; Phosphates/metabolism ; Uric Acid/metabolism ; Urinary Calculi/*diagnosis/metabolism ; },
abstract = {In the present paper was reported on the results of the topographic crystal-optic analysis of urinary calculi of 560 concrements. 59% of all urinary calculi had a different phase content in the nucleus and in the calyx. All frequent minerals of the urinary calculi could be proved in the nucleus of the calculus. It could be shown that nucleus and calyx may have a monomineral as well as a polymineral structure. It is referred to the importance of the topographic analysis of the urinary calculi for the metaphylaxis of the urolithiasis.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/metabolism
Humans
Methods
*Optics and Photonics
Oxalates/metabolism
Phosphates/metabolism
Uric Acid/metabolism
Urinary Calculi/*diagnosis/metabolism
RevDate: 2019-08-27
CmpDate: 1977-10-14
The kidney in disorders of calcium metabolism.
Contributions to nephrology, 7:114-127.
Additional Links: PMID-891199
Publisher:
PubMed:
Citation:
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@article {pmid891199,
year = {1977},
author = {Ritz, E and Massry, SG},
title = {The kidney in disorders of calcium metabolism.},
journal = {Contributions to nephrology},
volume = {7},
number = {},
pages = {114-127},
doi = {10.1159/000400119},
pmid = {891199},
issn = {0302-5144},
mesh = {Animals ; Calcium/urine ; Calcium Metabolism Disorders/complications/*physiopathology ; Female ; Humans ; Hypercalcemia/complications/physiopathology ; Hyperparathyroidism/complications/physiopathology ; Kidney/*physiopathology ; Kidney Calculi/etiology/physiopathology ; Kidney Diseases/etiology/metabolism ; Male ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Animals
Calcium/urine
Calcium Metabolism Disorders/complications/*physiopathology
Female
Humans
Hypercalcemia/complications/physiopathology
Hyperparathyroidism/complications/physiopathology
Kidney/*physiopathology
Kidney Calculi/etiology/physiopathology
Kidney Diseases/etiology/metabolism
Male
RevDate: 2018-02-15
CmpDate: 1977-10-14
Aspects of calcium metabolism in obstructive nephropathy: a metabolic and bone biopsy investigation.
Nephron, 19(1):32-43.
Additional Links: PMID-895959
Publisher:
PubMed:
Citation:
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@article {pmid895959,
year = {1977},
author = {Maschio, G and D'Angelo, A and Bonucci, E and Pagano, F and Ossi, E and Lupo, A and Valvo, E and Tessitore, N and Messa, P},
title = {Aspects of calcium metabolism in obstructive nephropathy: a metabolic and bone biopsy investigation.},
journal = {Nephron},
volume = {19},
number = {1},
pages = {32-43},
doi = {10.1159/000180863},
pmid = {895959},
issn = {1660-8151},
mesh = {Adult ; Bone Resorption ; Calcium/blood/*metabolism/urine ; Chronic Kidney Disease-Mineral and Bone Disorder/etiology ; Creatinine/urine ; Female ; Glomerular Filtration Rate ; Humans ; Hydroxyproline/urine ; Hyperparathyroidism, Secondary/etiology ; Ilium/pathology ; Kidney Calculi/*metabolism/pathology ; Male ; Middle Aged ; Osteomalacia/etiology ; Phosphates ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Bone Resorption
Calcium/blood/*metabolism/urine
Chronic Kidney Disease-Mineral and Bone Disorder/etiology
Creatinine/urine
Female
Glomerular Filtration Rate
Humans
Hydroxyproline/urine
Hyperparathyroidism, Secondary/etiology
Ilium/pathology
Kidney Calculi/*metabolism/pathology
Male
Middle Aged
Osteomalacia/etiology
Phosphates
RevDate: 2013-11-21
CmpDate: 1977-10-14
[Treatment of calcium oxalate calculi using Reducto-Special].
Wiener medizinische Wochenschrift (1946), 127(13):431-432.
Additional Links: PMID-899035
PubMed:
Citation:
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@article {pmid899035,
year = {1977},
author = {Swoboda, G},
title = {[Treatment of calcium oxalate calculi using Reducto-Special].},
journal = {Wiener medizinische Wochenschrift (1946)},
volume = {127},
number = {13},
pages = {431-432},
pmid = {899035},
issn = {0043-5341},
mesh = {Acrylates/therapeutic use ; Calcium/metabolism ; Cellulase/*therapeutic use ; Drug Combinations ; Humans ; Kidney Calculi/*drug therapy/metabolism ; Oxalates/metabolism ; Phosphates/therapeutic use ; },
}
MeSH Terms:
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Acrylates/therapeutic use
Calcium/metabolism
Cellulase/*therapeutic use
Drug Combinations
Humans
Kidney Calculi/*drug therapy/metabolism
Oxalates/metabolism
Phosphates/therapeutic use
RevDate: 2015-11-19
CmpDate: 1977-11-25
[Animal-experiment studies on the effect of magnesium and vitamin B 6 on calcium-oxalate nephrolithiasis].
Zeitschrift fur Urologie und Nephrologie, 70(6):419-427.
By chronic intoxication with ethylene glycol or acute intoxication by Na-glyoxalate in the animal experiment a Ca-oxalatenephrolithiasis could be produced. At this model the influence of magnesium, pyridoxine and phosphate was studied. The combination therapy of magnesium and vitamin B6 can completely prevent the formation of Ca-oxalate-microliths in the kidney. The production of a preparation with 200 mg MgO and 10 mg pyridoxine per tablet for the metaphylaxis of oxalate calculi is recommended.
Additional Links: PMID-906678
PubMed:
Citation:
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@article {pmid906678,
year = {1977},
author = {Schneider, HJ and Hesse, A and Berg, W and Kirsten, J and Nickel, H},
title = {[Animal-experiment studies on the effect of magnesium and vitamin B 6 on calcium-oxalate nephrolithiasis].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {70},
number = {6},
pages = {419-427},
pmid = {906678},
issn = {0044-3611},
mesh = {Animals ; Calcium/metabolism ; Chelating Agents/therapeutic use ; Female ; Kidney Calculi/*drug therapy/metabolism ; Magnesium/pharmacology/*therapeutic use ; Oxalates/metabolism ; Oxidative Phosphorylation/drug effects ; Pyridoxine/pharmacology/*therapeutic use ; Rabbits ; },
abstract = {By chronic intoxication with ethylene glycol or acute intoxication by Na-glyoxalate in the animal experiment a Ca-oxalatenephrolithiasis could be produced. At this model the influence of magnesium, pyridoxine and phosphate was studied. The combination therapy of magnesium and vitamin B6 can completely prevent the formation of Ca-oxalate-microliths in the kidney. The production of a preparation with 200 mg MgO and 10 mg pyridoxine per tablet for the metaphylaxis of oxalate calculi is recommended.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Animals
Calcium/metabolism
Chelating Agents/therapeutic use
Female
Kidney Calculi/*drug therapy/metabolism
Magnesium/pharmacology/*therapeutic use
Oxalates/metabolism
Oxidative Phosphorylation/drug effects
Pyridoxine/pharmacology/*therapeutic use
Rabbits
RevDate: 2013-11-21
CmpDate: 1977-11-25
[Etiopathogenic study of idiopathic hypercalciuria].
Annales de medecine interne, 128(5):421-430.
Additional Links: PMID-907255
PubMed:
Citation:
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@article {pmid907255,
year = {1977},
author = {Merceron, RE and Cavailloles, F and Delorme, ML and Courreges, JP and Raymond, JP and Klotz, HP},
title = {[Etiopathogenic study of idiopathic hypercalciuria].},
journal = {Annales de medecine interne},
volume = {128},
number = {5},
pages = {421-430},
pmid = {907255},
issn = {0003-410X},
mesh = {Adult ; Bone Resorption/complications ; Calcium/*urine ; Calcium Metabolism Disorders/*etiology ; Cataract/complications ; Female ; Humans ; Hydroxycholecalciferols/biosynthesis ; Kidney Calculi/*etiology ; Male ; Parathyroid Hormone/blood ; Phosphorus/blood ; Sex Factors ; Tetany/complications ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Bone Resorption/complications
Calcium/*urine
Calcium Metabolism Disorders/*etiology
Cataract/complications
Female
Humans
Hydroxycholecalciferols/biosynthesis
Kidney Calculi/*etiology
Male
Parathyroid Hormone/blood
Phosphorus/blood
Sex Factors
Tetany/complications
RevDate: 2013-11-21
CmpDate: 1978-01-27
Increased serum 1,25-dihydroxyvitamin D in idiopathic hypercalciuria.
The Journal of laboratory and clinical medicine, 90(6):955-962.
Additional Links: PMID-925484
PubMed:
Citation:
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@article {pmid925484,
year = {1977},
author = {Shen, FH and Baylink, DJ and Nielsen, RL and Sherrard, DJ and Ivey, JL and Haussler, MR},
title = {Increased serum 1,25-dihydroxyvitamin D in idiopathic hypercalciuria.},
journal = {The Journal of laboratory and clinical medicine},
volume = {90},
number = {6},
pages = {955-962},
pmid = {925484},
issn = {0022-2143},
mesh = {Adult ; Animals ; Calcium/blood/*urine ; Calcium Metabolism Disorders/*blood/etiology ; Dihydroxycholecalciferols/*blood ; Female ; Humans ; Hydroxycholecalciferols/*blood ; Kidney Calculi/*blood/etiology/urine ; Male ; Middle Aged ; Parathyroid Hormone/blood ; Phosphorus/blood ; Rats ; },
}
MeSH Terms:
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Adult
Animals
Calcium/blood/*urine
Calcium Metabolism Disorders/*blood/etiology
Dihydroxycholecalciferols/*blood
Female
Humans
Hydroxycholecalciferols/*blood
Kidney Calculi/*blood/etiology/urine
Male
Middle Aged
Parathyroid Hormone/blood
Phosphorus/blood
Rats
RevDate: 2013-11-21
CmpDate: 1978-01-27
[Active and recurrent kidney calculi. Metabolic findings and therapeutic consequences].
Tidsskrift for den Norske laegeforening : tidsskrift for praktisk medicin, ny raekke, 97(30):1551-1555.
Additional Links: PMID-929531
PubMed:
Citation:
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@article {pmid929531,
year = {1977},
author = {Jorgensen, H and Quist Paulsen, A and Berstad, A and Harbitz, TB},
title = {[Active and recurrent kidney calculi. Metabolic findings and therapeutic consequences].},
journal = {Tidsskrift for den Norske laegeforening : tidsskrift for praktisk medicin, ny raekke},
volume = {97},
number = {30},
pages = {1551-1555},
pmid = {929531},
issn = {0029-2001},
mesh = {Acidosis, Renal Tubular/metabolism ; Bendroflumethiazide/therapeutic use ; Calcium/*metabolism ; Female ; Humans ; Hyperparathyroidism/metabolism ; Kidney Calculi/drug therapy/*metabolism ; Kidney Tubules/metabolism ; Male ; Medullary Sponge Kidney/metabolism ; Parathyroid Hormone/blood ; Phosphates/*metabolism ; Recurrence ; Urinary Tract Infections/metabolism ; },
}
MeSH Terms:
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Acidosis, Renal Tubular/metabolism
Bendroflumethiazide/therapeutic use
Calcium/*metabolism
Female
Humans
Hyperparathyroidism/metabolism
Kidney Calculi/drug therapy/*metabolism
Kidney Tubules/metabolism
Male
Medullary Sponge Kidney/metabolism
Parathyroid Hormone/blood
Phosphates/*metabolism
Recurrence
Urinary Tract Infections/metabolism
RevDate: 2013-11-21
CmpDate: 1976-10-02
[Metabolic disorders in patients with urolithiasis].
Zeitschrift fur Urologie und Nephrologie, 69(4):295-299.
Additional Links: PMID-952104
PubMed:
Citation:
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@article {pmid952104,
year = {1976},
author = {Zechner, O},
title = {[Metabolic disorders in patients with urolithiasis].},
journal = {Zeitschrift fur Urologie und Nephrologie},
volume = {69},
number = {4},
pages = {295-299},
pmid = {952104},
issn = {0044-3611},
mesh = {Calcium/metabolism ; Cystinuria/etiology ; Humans ; Phosphates/metabolism ; Purines/metabolism ; Urinary Calculi/complications/*metabolism ; },
}
MeSH Terms:
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hide MeSH Terms
Calcium/metabolism
Cystinuria/etiology
Humans
Phosphates/metabolism
Purines/metabolism
Urinary Calculi/complications/*metabolism
RevDate: 2019-10-21
CmpDate: 1976-11-01
The effect of magnesium and fluoride on nephrocalcinosis and aortic calcification in rats given high sucrose diets with added phospnates.
Calcified tissue research, 20(3):291-302.
The study was conducted to observe in rats the possible modification of ectopic calcification by magnesium-orthophosphate-fluoride combinations, used as additives of diet for reduction of the cariogenicity of the sucrose. In rats, fed low magnesium diets, extra dietary orthophosphate (2%) considerably elevated the calcification of kidneys. Further additions of magnesium and fluoride partially reduced this adverse effect of phosphate. While the calcium content of the aorta in rats, fed low magnesium-high phosphate diet, was considerably elevated, the further addition of magnesium (40 ppm) partially reduced the calcifying effect of phosphate in aorta. Fluoride (15 ppm) together with magnesium (40 ppm) completely reduced it. The appearance of renal calculi caused by a low magnesium diet or by extra phosphate were similar according to light and electron microscopy except for the larger size in the latter case and occasional extratubular calculi found in groups with high phosphate-low magnesium and high phosphate with added magnesium diets.
Additional Links: PMID-953785
PubMed:
Citation:
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@article {pmid953785,
year = {1976},
author = {Luoma, H and Nuuja, T and Collan, Y and Nummikoski, P},
title = {The effect of magnesium and fluoride on nephrocalcinosis and aortic calcification in rats given high sucrose diets with added phospnates.},
journal = {Calcified tissue research},
volume = {20},
number = {3},
pages = {291-302},
pmid = {953785},
issn = {0008-0594},
mesh = {Animals ; Aorta/drug effects ; Calcinosis/chemically induced/*prevention & control ; Calcium/metabolism ; *Cariostatic Agents/adverse effects ; *Dietary Carbohydrates ; Female ; Fluorides/*pharmacology ; Food Additives ; Magnesium/*pharmacology ; Male ; Nephrocalcinosis/prevention & control ; Phosphates/*pharmacology ; Potassium/metabolism ; Rats ; *Sucrose ; },
abstract = {The study was conducted to observe in rats the possible modification of ectopic calcification by magnesium-orthophosphate-fluoride combinations, used as additives of diet for reduction of the cariogenicity of the sucrose. In rats, fed low magnesium diets, extra dietary orthophosphate (2%) considerably elevated the calcification of kidneys. Further additions of magnesium and fluoride partially reduced this adverse effect of phosphate. While the calcium content of the aorta in rats, fed low magnesium-high phosphate diet, was considerably elevated, the further addition of magnesium (40 ppm) partially reduced the calcifying effect of phosphate in aorta. Fluoride (15 ppm) together with magnesium (40 ppm) completely reduced it. The appearance of renal calculi caused by a low magnesium diet or by extra phosphate were similar according to light and electron microscopy except for the larger size in the latter case and occasional extratubular calculi found in groups with high phosphate-low magnesium and high phosphate with added magnesium diets.},
}
MeSH Terms:
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hide MeSH Terms
Animals
Aorta/drug effects
Calcinosis/chemically induced/*prevention & control
Calcium/metabolism
*Cariostatic Agents/adverse effects
*Dietary Carbohydrates
Female
Fluorides/*pharmacology
Food Additives
Magnesium/*pharmacology
Male
Nephrocalcinosis/prevention & control
Phosphates/*pharmacology
Potassium/metabolism
Rats
*Sucrose
RevDate: 2013-11-21
CmpDate: 1976-11-01
[Urolithiasis and mineral metabolism].
Hippokrates, 47(1):53-65.
Additional Links: PMID-955945
PubMed:
Citation:
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@article {pmid955945,
year = {1976},
author = {Schwille, PO},
title = {[Urolithiasis and mineral metabolism].},
journal = {Hippokrates},
volume = {47},
number = {1},
pages = {53-65},
pmid = {955945},
issn = {0018-2001},
mesh = {Acidosis, Renal Tubular/complications ; Calcium/*metabolism ; Cystine/*metabolism ; Hypercalcemia/complications ; Hyperparathyroidism/complications ; Iatrogenic Disease ; Kidney Calculi/*metabolism ; Phosphates/*metabolism ; Urea/*metabolism ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Acidosis, Renal Tubular/complications
Calcium/*metabolism
Cystine/*metabolism
Hypercalcemia/complications
Hyperparathyroidism/complications
Iatrogenic Disease
Kidney Calculi/*metabolism
Phosphates/*metabolism
Urea/*metabolism
RevDate: 2013-11-21
CmpDate: 1976-12-03
[Report on a symposium on the treatment of Ca-containing urinary calculi using ion exchangers, Munich, May 8, 1976].
Der Urologe. Ausg. A, 15(5):262.
Additional Links: PMID-973283
PubMed:
Citation:
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@article {pmid973283,
year = {1976},
author = {Schmiedt, E},
title = {[Report on a symposium on the treatment of Ca-containing urinary calculi using ion exchangers, Munich, May 8, 1976].},
journal = {Der Urologe. Ausg. A},
volume = {15},
number = {5},
pages = {262},
pmid = {973283},
issn = {0340-2592},
mesh = {Animals ; Calcium/*metabolism ; Cation Exchange Resins/*therapeutic use ; Humans ; Ion Exchange Resins/*therapeutic use ; Urinary Calculi/*drug therapy/metabolism/prevention & control ; },
}
MeSH Terms:
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hide MeSH Terms
Animals
Calcium/*metabolism
Cation Exchange Resins/*therapeutic use
Humans
Ion Exchange Resins/*therapeutic use
Urinary Calculi/*drug therapy/metabolism/prevention & control
RevDate: 2006-11-15
CmpDate: 1977-01-29
[Hypercalciuric and hyperparathyroid renal lithiasis].
Minerva medica, 67(55):3573-3581.
Idiopathic hypercalciuria was noted in 10% of a series of 1635 subjects with renal lithiasis. Eight-day administration of thiazide diuretics as a test for the discovery of latent hyperparathyroidism in idiopathic hypercalciuria is described. In 6 cases diagnosed in this way, surgery disclosed the presence of a parathyroid adenoma. Resection was followed by persistent hypercalciuria and, in some instances, renal lithiasis activity. The pathogenesis of associations of these frequently observed diseases is examined.
Additional Links: PMID-995303
PubMed:
Citation:
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@article {pmid995303,
year = {1976},
author = {Rapado, A and de la Calle, H and Castrillo, JM and Mancha, A and Traba, ML and Cifuentes Delatte, L},
title = {[Hypercalciuric and hyperparathyroid renal lithiasis].},
journal = {Minerva medica},
volume = {67},
number = {55},
pages = {3573-3581},
pmid = {995303},
issn = {0026-4806},
mesh = {Adenoma/complications ; Adult ; Aged ; Calcium Metabolism Disorders/complications/*etiology ; Female ; Humans ; Hyperparathyroidism/*complications ; Kidney Calculi/*etiology ; Male ; Middle Aged ; Parathyroid Neoplasms/complications ; },
abstract = {Idiopathic hypercalciuria was noted in 10% of a series of 1635 subjects with renal lithiasis. Eight-day administration of thiazide diuretics as a test for the discovery of latent hyperparathyroidism in idiopathic hypercalciuria is described. In 6 cases diagnosed in this way, surgery disclosed the presence of a parathyroid adenoma. Resection was followed by persistent hypercalciuria and, in some instances, renal lithiasis activity. The pathogenesis of associations of these frequently observed diseases is examined.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adenoma/complications
Adult
Aged
Calcium Metabolism Disorders/complications/*etiology
Female
Humans
Hyperparathyroidism/*complications
Kidney Calculi/*etiology
Male
Middle Aged
Parathyroid Neoplasms/complications
RevDate: 2017-11-16
CmpDate: 1975-08-26
Mineral interrelationships in ovine nutrition.
Journal of the American Veterinary Medical Association, 166(3):264-268.
Additional Links: PMID-1094014
PubMed:
Citation:
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@article {pmid1094014,
year = {1975},
author = {Pope, AL},
title = {Mineral interrelationships in ovine nutrition.},
journal = {Journal of the American Veterinary Medical Association},
volume = {166},
number = {3},
pages = {264-268},
pmid = {1094014},
issn = {0003-1488},
mesh = {Air Pollution ; *Animal Nutritional Physiological Phenomena ; Animals ; Calcium/metabolism ; Copper/metabolism/toxicity ; Magnesium Deficiency/veterinary ; Minerals/*metabolism/toxicity ; Molybdenum/metabolism/toxicity ; Nitrogen/metabolism ; Nutritional Requirements ; Phosphorus/metabolism ; Potassium/metabolism ; Selenium/metabolism ; Sheep/*metabolism ; Sheep Diseases/chemically induced ; Soil ; Sulfur/metabolism ; Urinary Calculi/veterinary ; Vitamin E/metabolism ; Zinc/metabolism ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Air Pollution
*Animal Nutritional Physiological Phenomena
Animals
Calcium/metabolism
Copper/metabolism/toxicity
Magnesium Deficiency/veterinary
Minerals/*metabolism/toxicity
Molybdenum/metabolism/toxicity
Nitrogen/metabolism
Nutritional Requirements
Phosphorus/metabolism
Potassium/metabolism
Selenium/metabolism
Sheep/*metabolism
Sheep Diseases/chemically induced
Soil
Sulfur/metabolism
Urinary Calculi/veterinary
Vitamin E/metabolism
Zinc/metabolism
RevDate: 2019-12-10
CmpDate: 1975-08-29
[A case of sarcoidosis with simultaneous primary hyperparathyroidism, coincedence or consequnce?].
Medizinische Klinik, 70(20):904-908.
We have reported of a case of generalised sarcoidosis with primary hyperparathyroidism. A pathogenetic relation between sarcoidosis associated with hypercalcemia and the development of parathyroid adenoma will be discussed and a causal connection will be proposed. According to our hypothesis every primary hyperparathyroidism could have developed from regulatory hyperfunction. This is illustrated by sarcoidosis with hypercalcemia and hypercalcuria. In this case a disturbance in vitamin D dependent calcium metabolism induces hyperplasia of the parathyroid which later leads to the development of a parathyroid adenoma. In addition a review of literature describing similar cases is given.
Additional Links: PMID-1094256
PubMed:
Citation:
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@article {pmid1094256,
year = {1975},
author = {Hartmann, F and Lehmann, H},
title = {[A case of sarcoidosis with simultaneous primary hyperparathyroidism, coincedence or consequnce?].},
journal = {Medizinische Klinik},
volume = {70},
number = {20},
pages = {904-908},
pmid = {1094256},
issn = {0025-8458},
mesh = {Adenoma/complications/etiology ; Adult ; Bibliographies as Topic ; Calcium/urine ; Calcium Metabolism Disorders/complications ; Diagnosis, Differential ; Humans ; Hypercalcemia/complications/*etiology ; Hyperparathyroidism/blood/*complications/etiology ; Hyperplasia/etiology ; Kidney Calculi/complications ; Male ; Parathyroid Neoplasms/complications/etiology ; Sarcoidosis/*complications/diagnosis ; Vitamin D/metabolism ; },
abstract = {We have reported of a case of generalised sarcoidosis with primary hyperparathyroidism. A pathogenetic relation between sarcoidosis associated with hypercalcemia and the development of parathyroid adenoma will be discussed and a causal connection will be proposed. According to our hypothesis every primary hyperparathyroidism could have developed from regulatory hyperfunction. This is illustrated by sarcoidosis with hypercalcemia and hypercalcuria. In this case a disturbance in vitamin D dependent calcium metabolism induces hyperplasia of the parathyroid which later leads to the development of a parathyroid adenoma. In addition a review of literature describing similar cases is given.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adenoma/complications/etiology
Adult
Bibliographies as Topic
Calcium/urine
Calcium Metabolism Disorders/complications
Diagnosis, Differential
Humans
Hypercalcemia/complications/*etiology
Hyperparathyroidism/blood/*complications/etiology
Hyperplasia/etiology
Kidney Calculi/complications
Male
Parathyroid Neoplasms/complications/etiology
Sarcoidosis/*complications/diagnosis
Vitamin D/metabolism
RevDate: 2013-11-21
CmpDate: 1976-02-21
Calcium and skeletal metabolism.
The Western journal of medicine, 123(6):447-458.
Additional Links: PMID-1105981
Full Text:
PubMed:
Citation:
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@article {pmid1105981,
year = {1975},
author = {Deftos, LJ and Roos, BA and Parthemore, JG},
title = {Calcium and skeletal metabolism.},
journal = {The Western journal of medicine},
volume = {123},
number = {6},
pages = {447-458},
pmid = {1105981},
issn = {0093-0415},
mesh = {Adult ; Bone Diseases/physiopathology ; Bone and Bones/*metabolism ; Calcitonin/metabolism/physiology ; Calcium/*metabolism ; Child ; Humans ; Hypercalcemia/etiology ; Hyperparathyroidism/complications/metabolism ; Osteitis Deformans/diagnosis ; Osteoporosis/etiology ; Parathyroid Hormone/metabolism/physiology ; Thyroid Neoplasms/diagnosis ; Urinary Calculi/etiology ; Vitamin D/physiology ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Bone Diseases/physiopathology
Bone and Bones/*metabolism
Calcitonin/metabolism/physiology
Calcium/*metabolism
Child
Humans
Hypercalcemia/etiology
Hyperparathyroidism/complications/metabolism
Osteitis Deformans/diagnosis
Osteoporosis/etiology
Parathyroid Hormone/metabolism/physiology
Thyroid Neoplasms/diagnosis
Urinary Calculi/etiology
Vitamin D/physiology
RevDate: 2019-07-06
CmpDate: 1975-06-21
A study of the renal handling and intestinal absorption of dibasic amino acids in a patient with genotype +/11 heterozygous cystinuria and idiopathic hypercalcuria.
Clinica chimica acta; international journal of clinical chemistry, 59(2):195-201.
A case is reported of a patient with idiopathic hypercalcuria who was referred for investigation of renal calculi. Studies of urinary amino acid excretion in the patient and all living members of his family, as well as studies of endogenous renal clearances of dibasic amino acids and cystine in the patient and his daughter, indicated that the patient had genotype +/11 heterozygous cystinuria. Intestinal perfusion studies showed however that the patient was able to absorb L-lysine, L-arginine normally from the jejunum and ileum. This suggests that a difference exists between renal and intestinal handling of lysine and arginine in cystinuria.
Additional Links: PMID-1120363
Publisher:
PubMed:
Citation:
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@article {pmid1120363,
year = {1975},
author = {Silk, DB and Perrett, D and Clark, ML and Stephens, AD and Scowen, EF},
title = {A study of the renal handling and intestinal absorption of dibasic amino acids in a patient with genotype +/11 heterozygous cystinuria and idiopathic hypercalcuria.},
journal = {Clinica chimica acta; international journal of clinical chemistry},
volume = {59},
number = {2},
pages = {195-201},
doi = {10.1016/0009-8981(75)90029-7},
pmid = {1120363},
issn = {0009-8981},
mesh = {Adult ; Amino Acids, Diamino/*metabolism ; Calcium/*metabolism/urine ; Cystinuria/*metabolism ; Genotype ; Heterozygote ; Humans ; Ileum/metabolism ; *Intestinal Absorption ; Jejunum/metabolism ; Kidney/*metabolism ; Kidney Calculi ; Male ; Metal Metabolism, Inborn Errors/*metabolism ; },
abstract = {A case is reported of a patient with idiopathic hypercalcuria who was referred for investigation of renal calculi. Studies of urinary amino acid excretion in the patient and all living members of his family, as well as studies of endogenous renal clearances of dibasic amino acids and cystine in the patient and his daughter, indicated that the patient had genotype +/11 heterozygous cystinuria. Intestinal perfusion studies showed however that the patient was able to absorb L-lysine, L-arginine normally from the jejunum and ileum. This suggests that a difference exists between renal and intestinal handling of lysine and arginine in cystinuria.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adult
Amino Acids, Diamino/*metabolism
Calcium/*metabolism/urine
Cystinuria/*metabolism
Genotype
Heterozygote
Humans
Ileum/metabolism
*Intestinal Absorption
Jejunum/metabolism
Kidney/*metabolism
Kidney Calculi
Male
Metal Metabolism, Inborn Errors/*metabolism
RevDate: 2013-11-21
CmpDate: 1975-09-05
[Analysis of the submandibular gland calculi with regard to the presence of side elements by means of an electronic microanalyzer].
Ceskoslovenska otolaryngologie, 24(3):176-179.
Additional Links: PMID-1132019
PubMed:
Citation:
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@article {pmid1132019,
year = {1975},
author = {Hlavácek, V and Ulrichová, D},
title = {[Analysis of the submandibular gland calculi with regard to the presence of side elements by means of an electronic microanalyzer].},
journal = {Ceskoslovenska otolaryngologie},
volume = {24},
number = {3},
pages = {176-179},
pmid = {1132019},
issn = {0009-0603},
mesh = {Calcium/metabolism ; Electronics, Medical ; Humans ; Micromanipulation ; Phosphorus/metabolism ; Salivary Duct Calculi/*metabolism ; Submandibular Gland/*metabolism ; Sulfur/metabolism ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Calcium/metabolism
Electronics, Medical
Humans
Micromanipulation
Phosphorus/metabolism
Salivary Duct Calculi/*metabolism
Submandibular Gland/*metabolism
Sulfur/metabolism
RevDate: 2013-11-21
CmpDate: 1975-10-30
Bone metabolism in quadriplegia: dissociation between calciuria and hydroxyprolinuria.
Archives of physical medicine and rehabilitation, 56(8):327-332.
Relationships between duration of paralysis, recumbency, muscular activity, and urinary loss of calcium (Cau), phosphorus (Pu), and hydroxyproline (OHPu) were studied daily in 32 traumatic quadriplegic patients during comprehensive rehabilitation and randomly in those readmitted for treatment. Within days after onset, quadriplegic patients had increases in Pu, then OHPu, and finally, Cau. Patients actively engaged in rehabilitation exercises showed a steady fall in OHPu, whereas CAU remained high for up to 18 months. Patients paralyzed for over 18 months (late) had low Cau, Pu and OHPu; but if the patients were kept in bed, OHPu increased rapidly. Patients with early quadriplegia have an increased bone remodeling, suggesting that the excess OHPu and Cau are derived from resorbed bone. Patients with late quadriplegia have little bone remodeling, therefore the excess OHPu occurring during recumbency may be derived from the resorption of new collagen produced during the removal of weight bearing without further change in muscular activity. Resorbed new collagen is excreted in part as large, OHP-containing polypeptides; these were found in the urine of quadriplegic patients, and therefore were present in blood and may play a role in initiating ectopic bone and renal calculi. The presence of OHP in 11 bladder calculi from quadriplegic patients tends to support this hypothesis. These studies indicate that muscular activity and weight bearing influence the bone metabolism of quadriplegic patients and suggest that the presence in body fluids of increased catabolic products from bone may have a role in bone-related complications.
Additional Links: PMID-1156124
PubMed:
Citation:
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@article {pmid1156124,
year = {1975},
author = {Claus-Walker, J and Spencer, WA and Carter, RE and Halstead, LS and Meier, RH and Campos, RJ},
title = {Bone metabolism in quadriplegia: dissociation between calciuria and hydroxyprolinuria.},
journal = {Archives of physical medicine and rehabilitation},
volume = {56},
number = {8},
pages = {327-332},
pmid = {1156124},
issn = {0003-9993},
mesh = {Adolescent ; Adult ; Bone and Bones/*metabolism ; Calcium/*urine ; Exercise Therapy ; Female ; Humans ; Hydroxyproline/*urine ; Male ; Middle Aged ; Phosphorus/urine ; Quadriplegia/*metabolism ; Regression Analysis ; Time Factors ; },
abstract = {Relationships between duration of paralysis, recumbency, muscular activity, and urinary loss of calcium (Cau), phosphorus (Pu), and hydroxyproline (OHPu) were studied daily in 32 traumatic quadriplegic patients during comprehensive rehabilitation and randomly in those readmitted for treatment. Within days after onset, quadriplegic patients had increases in Pu, then OHPu, and finally, Cau. Patients actively engaged in rehabilitation exercises showed a steady fall in OHPu, whereas CAU remained high for up to 18 months. Patients paralyzed for over 18 months (late) had low Cau, Pu and OHPu; but if the patients were kept in bed, OHPu increased rapidly. Patients with early quadriplegia have an increased bone remodeling, suggesting that the excess OHPu and Cau are derived from resorbed bone. Patients with late quadriplegia have little bone remodeling, therefore the excess OHPu occurring during recumbency may be derived from the resorption of new collagen produced during the removal of weight bearing without further change in muscular activity. Resorbed new collagen is excreted in part as large, OHP-containing polypeptides; these were found in the urine of quadriplegic patients, and therefore were present in blood and may play a role in initiating ectopic bone and renal calculi. The presence of OHP in 11 bladder calculi from quadriplegic patients tends to support this hypothesis. These studies indicate that muscular activity and weight bearing influence the bone metabolism of quadriplegic patients and suggest that the presence in body fluids of increased catabolic products from bone may have a role in bone-related complications.},
}
MeSH Terms:
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hide MeSH Terms
Adolescent
Adult
Bone and Bones/*metabolism
Calcium/*urine
Exercise Therapy
Female
Humans
Hydroxyproline/*urine
Male
Middle Aged
Phosphorus/urine
Quadriplegia/*metabolism
Regression Analysis
Time Factors
RevDate: 2016-11-23
CmpDate: 1975-11-22
[Liquid renal calculosis].
Minerva urologica, 27(4):158-163.
Additional Links: PMID-1160860
PubMed:
Citation:
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@article {pmid1160860,
year = {1975},
author = {Piazza, G and Macchi, C and Cosciani, S and D'Arcais, R},
title = {[Liquid renal calculosis].},
journal = {Minerva urologica},
volume = {27},
number = {4},
pages = {158-163},
pmid = {1160860},
issn = {0026-4989},
mesh = {Adult ; Calcium/metabolism ; Humans ; Kidney Calculi/*diagnostic imaging/metabolism/surgery ; Male ; Radiography ; },
}
MeSH Terms:
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Adult
Calcium/metabolism
Humans
Kidney Calculi/*diagnostic imaging/metabolism/surgery
Male
Radiography
RevDate: 2013-11-21
CmpDate: 1975-11-22
[A special cause of urinary lithiasis: dysfunction of the terminal ileum].
La Nouvelle presse medicale.
Additional Links: PMID-1161461
PubMed:
Citation:
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@article {pmid1161461,
year = {1975},
author = {Cassan, P and Baglin, A},
title = {[A special cause of urinary lithiasis: dysfunction of the terminal ileum].},
journal = {La Nouvelle presse medicale},
volume = {},
number = {23},
pages = {1729-1731},
pmid = {1161461},
issn = {0301-1518},
mesh = {Bile Acids and Salts/metabolism ; Calcium/metabolism ; Crohn Disease/*complications ; Glycine/metabolism ; Glyoxylates/metabolism ; Humans ; Ileum/*physiopathology ; Intestinal Absorption ; Malabsorption Syndromes/*complications ; Oxalates/blood/metabolism ; Urinary Calculi/*etiology/prevention & control ; },
}
MeSH Terms:
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Bile Acids and Salts/metabolism
Calcium/metabolism
Crohn Disease/*complications
Glycine/metabolism
Glyoxylates/metabolism
Humans
Ileum/*physiopathology
Intestinal Absorption
Malabsorption Syndromes/*complications
Oxalates/blood/metabolism
Urinary Calculi/*etiology/prevention & control
RevDate: 2013-11-21
CmpDate: 1976-04-02
Proceedings: Acid and alkaline urinary inorganic pyrophosphatase in the formation of calcium-containing urinary calculi.
Israel journal of medical sciences, 11(11):1226-1227.
Additional Links: PMID-1205831
PubMed:
Citation:
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@article {pmid1205831,
year = {1975},
author = {Eisenkraft, C and Gaist, L and Rubinstein, J and Menache, R},
title = {Proceedings: Acid and alkaline urinary inorganic pyrophosphatase in the formation of calcium-containing urinary calculi.},
journal = {Israel journal of medical sciences},
volume = {11},
number = {11},
pages = {1226-1227},
pmid = {1205831},
issn = {0021-2180},
mesh = {Calcium/*metabolism ; Humans ; Pyrophosphatases/*physiology ; Urinary Calculi/*enzymology ; },
}
MeSH Terms:
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Calcium/*metabolism
Humans
Pyrophosphatases/*physiology
Urinary Calculi/*enzymology
RevDate: 2006-11-15
CmpDate: 1976-03-15
[Aspects of phosphorus-calcium metabolism in obstructive nephropathy].
Minerva nefrologica, 22(1):38-44.
Additional Links: PMID-1207972
PubMed:
Citation:
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@article {pmid1207972,
year = {1975},
author = {Maschio, G and D'Angelo, A and Ossi, E and Lupo, A and Valvo, E and Bonucci, E and Pagano, F and D'Amico, C and Passerini, G and Pegoraro, V},
title = {[Aspects of phosphorus-calcium metabolism in obstructive nephropathy].},
journal = {Minerva nefrologica},
volume = {22},
number = {1},
pages = {38-44},
pmid = {1207972},
issn = {0026-4873},
mesh = {Calcium Metabolism Disorders/*complications ; Glomerulonephritis/complications ; Humans ; Hyperparathyroidism/etiology ; Kidney Calculi/*complications/etiology ; Kidney Failure, Chronic/etiology ; Osteomalacia/etiology ; Phosphorus Metabolism Disorders/*complications ; Pyelonephritis/complications ; },
}
MeSH Terms:
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hide MeSH Terms
Calcium Metabolism Disorders/*complications
Glomerulonephritis/complications
Humans
Hyperparathyroidism/etiology
Kidney Calculi/*complications/etiology
Kidney Failure, Chronic/etiology
Osteomalacia/etiology
Phosphorus Metabolism Disorders/*complications
Pyelonephritis/complications
RevDate: 2013-11-21
CmpDate: 1976-05-25
[Evaluation of hyperparathyroidism and recurrent renal lithiasis].
Journal d'urologie et de nephrologie, 81(10-11):824-830.
Additional Links: PMID-1219134
PubMed:
Citation:
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@article {pmid1219134,
year = {1975},
author = {Bernheim, J and Pozet, N and Zech, P and Archimbaud, JP and Dubernard, JM and Traeger, J and Perrin, J},
title = {[Evaluation of hyperparathyroidism and recurrent renal lithiasis].},
journal = {Journal d'urologie et de nephrologie},
volume = {81},
number = {10-11},
pages = {824-830},
pmid = {1219134},
issn = {0021-8200},
mesh = {Adult ; Calcium/*metabolism ; Humans ; Hyperparathyroidism/*complications/diagnosis ; Kidney Calculi/*etiology ; Parathyroid Hormone/*blood ; Phosphates/*metabolism ; },
}
MeSH Terms:
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hide MeSH Terms
Adult
Calcium/*metabolism
Humans
Hyperparathyroidism/*complications/diagnosis
Kidney Calculi/*etiology
Parathyroid Hormone/*blood
Phosphates/*metabolism
RevDate: 2016-11-23
CmpDate: 1976-07-06
[Primary hyperparathyroidism. 50 personal cases].
Revue du rhumatisme et des maladies osteo-articulaires, 42(12):747-758.
On the basis of a study of a series of 50 personal cases, the results of which are reported, and of a series of 2800 cases from the literature, the authors describe certain radiological and clinical aspects of primary hyperparathyroidism and the conditions under which this disease may appear. They emphasize the value of the different laboratory investigations for determining disturbances in phosphorus-calcium metabolism and of the study of bone histology, particularly the quantitative study of iliac bone biopsies. They discuss the various aspects of their methods of surgical treatment (pre-operative location of the tumour, surgical problems, early and later sequelae) and finally describe the indications for surgery.
Additional Links: PMID-1224160
PubMed:
Citation:
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@article {pmid1224160,
year = {1975},
author = {Deplante, JP and Daumont, A and Bouvier, M and Lejeune, E},
title = {[Primary hyperparathyroidism. 50 personal cases].},
journal = {Revue du rhumatisme et des maladies osteo-articulaires},
volume = {42},
number = {12},
pages = {747-758},
pmid = {1224160},
issn = {0035-2659},
mesh = {Adenoma/surgery ; Adult ; Calcium/blood ; Chondrocalcinosis/etiology ; Elbow/diagnostic imaging ; Female ; Humans ; *Hyperparathyroidism ; Ilium/pathology ; Kidney Calculi/etiology ; Male ; Middle Aged ; Parathyroid Glands/surgery ; Pelvis/diagnostic imaging ; Radiography ; Skull/diagnostic imaging ; },
abstract = {On the basis of a study of a series of 50 personal cases, the results of which are reported, and of a series of 2800 cases from the literature, the authors describe certain radiological and clinical aspects of primary hyperparathyroidism and the conditions under which this disease may appear. They emphasize the value of the different laboratory investigations for determining disturbances in phosphorus-calcium metabolism and of the study of bone histology, particularly the quantitative study of iliac bone biopsies. They discuss the various aspects of their methods of surgical treatment (pre-operative location of the tumour, surgical problems, early and later sequelae) and finally describe the indications for surgery.},
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Adenoma/surgery
Adult
Calcium/blood
Chondrocalcinosis/etiology
Elbow/diagnostic imaging
Female
Humans
*Hyperparathyroidism
Ilium/pathology
Kidney Calculi/etiology
Male
Middle Aged
Parathyroid Glands/surgery
Pelvis/diagnostic imaging
Radiography
Skull/diagnostic imaging
RevDate: 2013-11-21
CmpDate: 1976-08-02
[The excretion of oxalates in urine following oral administration of oxalic acid and magnesium].
Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten, 35(5):241-245.
Additional Links: PMID-1225513
PubMed:
Citation:
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@article {pmid1225513,
year = {1975},
author = {Berg, W and Schneider, HJ and Vogel, E},
title = {[The excretion of oxalates in urine following oral administration of oxalic acid and magnesium].},
journal = {Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten},
volume = {35},
number = {5},
pages = {241-245},
pmid = {1225513},
issn = {0012-1053},
mesh = {Administration, Oral ; Calcium/metabolism ; Circadian Rhythm ; Humans ; Magnesium/*administration & dosage/therapeutic use ; Oxalates/administration & dosage/*urine ; Urinary Calculi/drug therapy ; Vegetables ; },
}
MeSH Terms:
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Administration, Oral
Calcium/metabolism
Circadian Rhythm
Humans
Magnesium/*administration & dosage/therapeutic use
Oxalates/administration & dosage/*urine
Urinary Calculi/drug therapy
Vegetables
RevDate: 2013-11-21
CmpDate: 1976-10-02
[Disorders of electrolyte and acid-base metabolism in children after nephrectomy for nephrolithiasis].
Urologiia i nefrologiia.
Additional Links: PMID-1231120
PubMed:
Citation:
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@article {pmid1231120,
year = {1975},
author = {Shimonko, IT},
title = {[Disorders of electrolyte and acid-base metabolism in children after nephrectomy for nephrolithiasis].},
journal = {Urologiia i nefrologiia},
volume = {},
number = {5},
pages = {7-10},
pmid = {1231120},
issn = {0042-1154},
mesh = {Acid-Base Imbalance/*etiology ; Adolescent ; Calcium/metabolism ; Child ; Female ; Follow-Up Studies ; Humans ; Kidney/*metabolism ; Male ; *Nephrectomy ; Postoperative Complications/*metabolism ; Potassium/metabolism ; Sodium/metabolism ; Urinary Calculi/surgery ; Water-Electrolyte Imbalance/*etiology ; },
}
MeSH Terms:
show MeSH Terms
hide MeSH Terms
Acid-Base Imbalance/*etiology
Adolescent
Calcium/metabolism
Child
Female
Follow-Up Studies
Humans
Kidney/*metabolism
Male
*Nephrectomy
Postoperative Complications/*metabolism
Potassium/metabolism
Sodium/metabolism
Urinary Calculi/surgery
Water-Electrolyte Imbalance/*etiology
RevDate: 2013-11-21
CmpDate: 1976-08-02
[Calcium, phosphorus and magnesium metabolism in patients with recurrent nephrolithiasis].
Polski przeglad chirurgiczny, 48(5):605-610.
Additional Links: PMID-1272989
PubMed:
Citation:
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@article {pmid1272989,
year = {1976},
author = {Kwias, Z and Twardosz, W},
title = {[Calcium, phosphorus and magnesium metabolism in patients with recurrent nephrolithiasis].},
journal = {Polski przeglad chirurgiczny},
volume = {48},
number = {5},
pages = {605-610},
pmid = {1272989},
issn = {0032-373X},
mesh = {Calcium/*metabolism ; Humans ; Hyperparathyroidism/diagnosis ; Kidney Calculi/*metabolism ; Magnesium/*metabolism ; Phosphorus/*metabolism ; Recurrence ; },
}
MeSH Terms:
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Calcium/*metabolism
Humans
Hyperparathyroidism/diagnosis
Kidney Calculi/*metabolism
Magnesium/*metabolism
Phosphorus/*metabolism
Recurrence
RevDate: 2013-11-21
CmpDate: 1976-08-02
Medical management of urolithiasis.
American family physician, 13(5):89-94.
Knowledge of the crystalline structure of the calculus provides the basis of the therapeutic plan. Laboratory evaluation depends heavily upon routine urinalysis. Assessment of renal function, serum calcium, phosphorus, uric acid and, in some cases electrolytes is usually indicated, as is urography. General principles of management include maintenance of an ample urine volume, eradication of infection and correction of any obstructing lesions or metabolic abnormalities. Specific antistone regimens are indicated for patients with recurrent urolithiasis.
Additional Links: PMID-1274811
PubMed:
Citation:
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@article {pmid1274811,
year = {1976},
author = {Bissada, NK and Redman, JF},
title = {Medical management of urolithiasis.},
journal = {American family physician},
volume = {13},
number = {5},
pages = {89-94},
pmid = {1274811},
issn = {0002-838X},
mesh = {Anti-Bacterial Agents/therapeutic use ; Calcium/metabolism ; Cystinuria/complications ; Diarrhea/chemically induced ; Female ; Humans ; Hydrochlorothiazide/adverse effects/therapeutic use ; Intestinal Diseases/complications ; Kidney Calculi/complications ; Male ; Oxalates ; Phosphates/adverse effects/therapeutic use ; Proteus Infections/drug therapy ; Uric Acid/metabolism ; Urinary Calculi/*drug therapy/etiology/metabolism ; },
abstract = {Knowledge of the crystalline structure of the calculus provides the basis of the therapeutic plan. Laboratory evaluation depends heavily upon routine urinalysis. Assessment of renal function, serum calcium, phosphorus, uric acid and, in some cases electrolytes is usually indicated, as is urography. General principles of management include maintenance of an ample urine volume, eradication of infection and correction of any obstructing lesions or metabolic abnormalities. Specific antistone regimens are indicated for patients with recurrent urolithiasis.},
}
MeSH Terms:
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Anti-Bacterial Agents/therapeutic use
Calcium/metabolism
Cystinuria/complications
Diarrhea/chemically induced
Female
Humans
Hydrochlorothiazide/adverse effects/therapeutic use
Intestinal Diseases/complications
Kidney Calculi/complications
Male
Oxalates
Phosphates/adverse effects/therapeutic use
Proteus Infections/drug therapy
Uric Acid/metabolism
Urinary Calculi/*drug therapy/etiology/metabolism
RevDate: 2013-11-21
CmpDate: 1992-11-05
[Hypercalciuria].
Vnitrni lekarstvi, 38(7):672-677.
Hypercalciuria is the cause of almost 20% of all secondary osteoporoses and in 23% these cases are associated with calcium urolithiasis. It is therefore necessary to search for these patients actively because their treatment with hydrochlorothiazide and amiloride is easy and highly effective. We must not be satisfied with the finding of hypercalciuria as the only cause of demineralization of bone, as several causes may combine in a single patient. Comprehensive treatment of osteopenia associated with hypercalciuria is relatively shorter and more successful than in other forms of secondary osteopenias.
Additional Links: PMID-1413570
PubMed:
Citation:
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@article {pmid1413570,
year = {1992},
author = {Kocián, J},
title = {[Hypercalciuria].},
journal = {Vnitrni lekarstvi},
volume = {38},
number = {7},
pages = {672-677},
pmid = {1413570},
issn = {0042-773X},
mesh = {Calcium/*urine ; Calcium Metabolism Disorders/complications/diagnosis/urine ; Female ; Humans ; Male ; Osteoporosis/complications ; Urinary Calculi/complications ; },
abstract = {Hypercalciuria is the cause of almost 20% of all secondary osteoporoses and in 23% these cases are associated with calcium urolithiasis. It is therefore necessary to search for these patients actively because their treatment with hydrochlorothiazide and amiloride is easy and highly effective. We must not be satisfied with the finding of hypercalciuria as the only cause of demineralization of bone, as several causes may combine in a single patient. Comprehensive treatment of osteopenia associated with hypercalciuria is relatively shorter and more successful than in other forms of secondary osteopenias.},
}
MeSH Terms:
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Calcium/*urine
Calcium Metabolism Disorders/complications/diagnosis/urine
Female
Humans
Male
Osteoporosis/complications
Urinary Calculi/complications
RevDate: 2013-11-21
CmpDate: 1993-02-02
[Influence of parathyroidectomy on the clinical course of primary hyperparathyroidism].
Revista clinica espanola, 191(7):385-392.
Additional Links: PMID-1475467
PubMed:
Citation:
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@article {pmid1475467,
year = {1992},
author = {Castrillo, JM},
title = {[Influence of parathyroidectomy on the clinical course of primary hyperparathyroidism].},
journal = {Revista clinica espanola},
volume = {191},
number = {7},
pages = {385-392},
pmid = {1475467},
issn = {0014-2565},
mesh = {Calcium/metabolism ; Chondrocalcinosis/etiology/therapy ; Female ; Follow-Up Studies ; Gastrointestinal Diseases/etiology/therapy ; Humans ; Hyperparathyroidism/complications/metabolism/*surgery ; Hypertension/etiology/therapy ; Intestinal Absorption ; Kidney Calculi/etiology/therapy ; Male ; Neuromuscular Diseases/etiology/therapy ; Osteitis Fibrosa Cystica/etiology/therapy ; *Parathyroidectomy ; Postoperative Complications ; Uric Acid/blood ; },
}
MeSH Terms:
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Calcium/metabolism
Chondrocalcinosis/etiology/therapy
Female
Follow-Up Studies
Gastrointestinal Diseases/etiology/therapy
Humans
Hyperparathyroidism/complications/metabolism/*surgery
Hypertension/etiology/therapy
Intestinal Absorption
Kidney Calculi/etiology/therapy
Male
Neuromuscular Diseases/etiology/therapy
Osteitis Fibrosa Cystica/etiology/therapy
*Parathyroidectomy
Postoperative Complications
Uric Acid/blood
RevDate: 2017-11-16
CmpDate: 1993-02-12
Effect of "Rosa Canina" infusion and magnesium on the urinary risk factors of calcium oxalate urolithiasis.
Planta medica, 58(6):509-512.
The effects on the calcium oxalate urolithiasis urinary risk factors of "Rosa Canina", in herb infusion form, and magnesium chloride have been studied using female Wistar rats under balanced dietary conditions. No significant effects on the volume of liquids drunk or on creatinine, phosphate, and oxalate urinary concentrations and excretions were observed. The herb infusion did not cause any diuretic effect. Calciuria decreased and citraturia increased when taking the herb infusion, and vice versa when taking magnesium chloride. Magnesium chloride decreased the urinary pH value, but this effect was not observed when magnesium chloride was administered with herb infusion. In conclusion, the same beneficial effects of the studied infusion herb on calcium oxalate urolithiasis urinary risk factors can be clearly detected. An interesting fact is that it seems that some possible effects depend on dietary components, thus, i.e., an increase in the urinary pH was only detected when the intake of the herb infusion was studied in a magnesium chloride-supplemented diet.
Additional Links: PMID-1484889
Publisher:
PubMed:
Citation:
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@article {pmid1484889,
year = {1992},
author = {Grases, F and Masárová, L and Costa-Bauzá, A and March, JG and Prieto, R and Tur, JA},
title = {Effect of "Rosa Canina" infusion and magnesium on the urinary risk factors of calcium oxalate urolithiasis.},
journal = {Planta medica},
volume = {58},
number = {6},
pages = {509-512},
doi = {10.1055/s-2006-961537},
pmid = {1484889},
issn = {0032-0943},
mesh = {Animals ; *Calcium Oxalate ; Female ; Magnesium/therapeutic use ; *Magnoliopsida ; Rats ; Rats, Wistar ; Risk Factors ; Urinary Calculi/chemistry/*prevention & control ; },
abstract = {The effects on the calcium oxalate urolithiasis urinary risk factors of "Rosa Canina", in herb infusion form, and magnesium chloride have been studied using female Wistar rats under balanced dietary conditions. No significant effects on the volume of liquids drunk or on creatinine, phosphate, and oxalate urinary concentrations and excretions were observed. The herb infusion did not cause any diuretic effect. Calciuria decreased and citraturia increased when taking the herb infusion, and vice versa when taking magnesium chloride. Magnesium chloride decreased the urinary pH value, but this effect was not observed when magnesium chloride was administered with herb infusion. In conclusion, the same beneficial effects of the studied infusion herb on calcium oxalate urolithiasis urinary risk factors can be clearly detected. An interesting fact is that it seems that some possible effects depend on dietary components, thus, i.e., an increase in the urinary pH was only detected when the intake of the herb infusion was studied in a magnesium chloride-supplemented diet.},
}
MeSH Terms:
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Animals
*Calcium Oxalate
Female
Magnesium/therapeutic use
*Magnoliopsida
Rats
Rats, Wistar
Risk Factors
Urinary Calculi/chemistry/*prevention & control
RevDate: 2022-03-21
CmpDate: 1992-09-10
[Inflammation and subcutaneous calcification of venous origins].
Phlebologie, 45(1):41-7; discussion 48-9.
There have been few descriptions up to now of calcifications in chronic venous insufficiency, other than in cases where venous insufficiency is complicated be severe trophic disorders and in particular ulcers. It was therefore felt to be of interest to assess the presence of calcifications in venous insufficiency without trophic disorders. This study was based upon 40 cases recruited in the phlebology out-patient clinic of the Notre Dame de Bon Secours Hospital. Calcifications of the lower limbs were found in 7 patients, either by palpation, routine X-rays or ultrasonography. The etiopathogenic mechanisms of this occurrence not having been elucidated, a number of hypotheses are put forward on the basis of acquired data concerning: the process of formation of ectopic calcifications, changes in subcutaneous tissue, the ultimate consequences of venous stasis and of raised venous pressure, due essentially to anoxia and inflammation. One hypothesis can thus be put forward: that of inflammation. The release of cells and mediators of inflammation, the production of free radicals, causing damage to the cells of connective tissue and to the organic framework (collagen fibres) and changes in the chemical environment could combine to result in the formation of calcifications in subcutaneous tissue. However, inflammation has not been proven to be the primary etiological factor.
Additional Links: PMID-1496031
PubMed:
Citation:
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@article {pmid1496031,
year = {1992},
author = {Piry, A and Vin, F and Allaert, FA},
title = {[Inflammation and subcutaneous calcification of venous origins].},
journal = {Phlebologie},
volume = {45},
number = {1},
pages = {41-7; discussion 48-9},
pmid = {1496031},
issn = {0031-8280},
mesh = {Adult ; Aged ; Aged, 80 and over ; Calcinosis/*etiology/metabolism/physiopathology ; Calcium/metabolism ; Calculi/etiology/metabolism/physiopathology ; Chronic Disease ; Female ; Humans ; Leg/blood supply ; Male ; Middle Aged ; Phlebitis/*complications/metabolism/physiopathology ; Phosphorus/metabolism ; Skin/*blood supply ; Venous Insufficiency/*etiology/metabolism/physiopathology ; },
abstract = {There have been few descriptions up to now of calcifications in chronic venous insufficiency, other than in cases where venous insufficiency is complicated be severe trophic disorders and in particular ulcers. It was therefore felt to be of interest to assess the presence of calcifications in venous insufficiency without trophic disorders. This study was based upon 40 cases recruited in the phlebology out-patient clinic of the Notre Dame de Bon Secours Hospital. Calcifications of the lower limbs were found in 7 patients, either by palpation, routine X-rays or ultrasonography. The etiopathogenic mechanisms of this occurrence not having been elucidated, a number of hypotheses are put forward on the basis of acquired data concerning: the process of formation of ectopic calcifications, changes in subcutaneous tissue, the ultimate consequences of venous stasis and of raised venous pressure, due essentially to anoxia and inflammation. One hypothesis can thus be put forward: that of inflammation. The release of cells and mediators of inflammation, the production of free radicals, causing damage to the cells of connective tissue and to the organic framework (collagen fibres) and changes in the chemical environment could combine to result in the formation of calcifications in subcutaneous tissue. However, inflammation has not been proven to be the primary etiological factor.},
}
MeSH Terms:
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Adult
Aged
Aged, 80 and over
Calcinosis/*etiology/metabolism/physiopathology
Calcium/metabolism
Calculi/etiology/metabolism/physiopathology
Chronic Disease
Female
Humans
Leg/blood supply
Male
Middle Aged
Phlebitis/*complications/metabolism/physiopathology
Phosphorus/metabolism
Skin/*blood supply
Venous Insufficiency/*etiology/metabolism/physiopathology
RevDate: 2013-11-21
CmpDate: 1992-06-11
[Idiopathic hypercalciuria and bone density].
Revue du rhumatisme et des maladies osteo-articulaires, 59(1):35-38.
A decrease in bone density of the spine has been reported in individuals with hypercalciuria and the finding of the latter in osteoporosis patients is not uncommon. We studied 21 men and 8 women (mean age 47 +/- 13) with idiopathic hypercalciuria (IHCU) defined by an urinary calcium of more than 7.5 mmol/24 h in men and 6.25 mmol/24 h in women. The duration of IHCU was 10 (+/-) 8 years. Among the 29 patients, 24 had one or more renal calculi. Twenty one had been treated, by low calcium diet only (and diuresis), combined with a thiazide diuretic, or sodium phytate, or phosphorus. Bone mineral content (BMC) was measured in the lumbar spine and the upper end of the femur using an ORIS ODC 200 densitometer and compared with 29 control subjects paired for age and sex. No difference was found between the two groups concerning BMC values in either the spine or the 3 femoral sites (neck, Ward, trochanter). BMC was not correlated with urinary calcium. Thus individuals with IHCU showed no decrease in their bone mass, among this group seen in a department of nephrology. The influence of the treatment of IHCU remains to be defined.
Additional Links: PMID-1579844
PubMed:
Citation:
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@article {pmid1579844,
year = {1992},
author = {Daragon, A and Dhib, M and Morin, JP and Godin, M and Fillastre, JP},
title = {[Idiopathic hypercalciuria and bone density].},
journal = {Revue du rhumatisme et des maladies osteo-articulaires},
volume = {59},
number = {1},
pages = {35-38},
pmid = {1579844},
issn = {0035-2659},
mesh = {Adult ; Bone Density/*physiology ; Calcium/*urine ; Calcium Metabolism Disorders/*physiopathology ; Female ; Femur Head/physiopathology ; Humans ; Lumbar Vertebrae ; Male ; Middle Aged ; Spine/physiopathology ; },
abstract = {A decrease in bone density of the spine has been reported in individuals with hypercalciuria and the finding of the latter in osteoporosis patients is not uncommon. We studied 21 men and 8 women (mean age 47 +/- 13) with idiopathic hypercalciuria (IHCU) defined by an urinary calcium of more than 7.5 mmol/24 h in men and 6.25 mmol/24 h in women. The duration of IHCU was 10 (+/-) 8 years. Among the 29 patients, 24 had one or more renal calculi. Twenty one had been treated, by low calcium diet only (and diuresis), combined with a thiazide diuretic, or sodium phytate, or phosphorus. Bone mineral content (BMC) was measured in the lumbar spine and the upper end of the femur using an ORIS ODC 200 densitometer and compared with 29 control subjects paired for age and sex. No difference was found between the two groups concerning BMC values in either the spine or the 3 femoral sites (neck, Ward, trochanter). BMC was not correlated with urinary calcium. Thus individuals with IHCU showed no decrease in their bone mass, among this group seen in a department of nephrology. The influence of the treatment of IHCU remains to be defined.},
}
MeSH Terms:
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Adult
Bone Density/*physiology
Calcium/*urine
Calcium Metabolism Disorders/*physiopathology
Female
Femur Head/physiopathology
Humans
Lumbar Vertebrae
Male
Middle Aged
Spine/physiopathology
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Robbins holds BS, MS, and PhD degrees in the life sciences. He served as a tenured faculty member in the Zoology and Biological Science departments at Michigan State University. He is currently exploring the intersection between genomics, microbial ecology, and biodiversity — an area that promises to transform our understanding of the biosphere.
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